Evidence map›Paper›PMID 32419121›Full record

ArticleNeurochemical research2020

HAP1 Modulates Epileptic Seizures by Regulating GABA

Rong Li, Bing Wu, Miaoqing He, Peng Zhang, Qinbin Zhang, Jing Deng, Jinxian Yuan, Yangmei Chen

Abstract read
PubMed Publisher
In one paragraph

Article in Neurochemical research, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
1.0field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Clptm1, a new target in suppressing epileptic seizure by regulating GABAThe Kaohsiung journal of medical sciences · 2023
    Article
  5. Article
  6. Article
  7. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 1 country.

Rong LiDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Bing WuDepartment of Neurology, Chongqing Key Laboratory of Neurology, First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Miaoqing HeCenter for Brain Disorders Research, Capital Medical University, Beijing, China.
Peng ZhangDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Qinbin ZhangDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Jing DengDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Jinxian YuanDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China.
Yangmei ChenDepartment of Neurology, Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400016, China. chenym1997@cqmu.edu.cn.
Chongqing Medical University · CNSecond Affiliated Hospital of Chongqing Medical University · CNChinese Institute for Brain Research · CNDalian Medical University · CN

Funding

National Natural Science Foundation of China 81771390
6 · The paper itself

Abstract

The number of γ-aminobutyric acid type A receptors (GABA

Indexed as

AdolescentAdultAnimalsChildChild, PreschoolEpilepsy, Temporal LobeFemaleGABA AntagonistsGene Knockout TechniquesHumansInhibitory Postsynaptic PotentialsMaleNerve Tissue ProteinsPentylenetetrazolePicrotoxinRats, Sprague-DawleyGABA AntagonistsGABRB2 protein, humanGabrb2 protein, ratHAP1 protein, humanHap1 protein, ratNerve Tissue ProteinsPentylenetetrazolePicrotoxinReceptors, GABA-AEpileptic seizureGABAArβ2/3HAP1PTZTLE

Identifiers

PMID32419121
OpenAlexW3025066523

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.