Evidence map›Paper›PMID 32404075›Full record

ArticleBMC public health2020

Association between dyslipidemia and blood lipids concentration with smoking habits in the Kurdish population of Iran.

Mehdi Moradinazar, Yahya Pasdar, Farid Najafi, Soodeh Shahsavari, Ebrahim Shakiba, Behrooz Hamzeh, Negin Fakhri

Open access · goldAbstract read
In one paragraph

Article in BMC public health, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed, 1 pooled it
4.3field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 1 synthesis or guideline pooled it, 59 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Mehdi MoradinazarBehavioral Disease Research Center, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Yahya PasdarNutritional Sciences Department, School of Public Health, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Farid NajafiResearch Center for Environmental Determinants of Health, School of Public Health, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Soodeh ShahsavariBehavioral Disease Research Center, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Ebrahim ShakibaBehavioral Disease Research Center, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Behrooz HamzehResearch Center for Environmental Determinants of Health, School of Public Health, Kermanshah University of Medical Sciences, Kermanshah, Iran.
Negin FakhriStudent's research committee, Faculty of Health, Kermanshah University of medical sciences, Kermanshah, Iran. n.fakhri94@yahoo.com.
Kermanshah University of Medical Sciences · IR

Funding

Kermanshah University of Medical Sciences 92472
6 · The paper itself

Abstract

backgroundSmoking is the most preventable cause of most chronic diseases such as cardiovascular disease (CVD). Dyslipidemia is also an important risk factor for CVD. Yet, research has provided contradicting findings regarding the association between smoking and blood lipids. This paper examines the relationship between dyslipidemia and smoking based on the results of a cross-sectional sample of a Kurdish population in western Iran.

methodsThis population-based study was derived from the recruitment phase of Ravansar Non-Communicable Disease (RaNCD) cohort study. Logistic regression model adjusted by confounding variables was used to determine the relationship between smoking and blood lipid components. In addition, dose-response relationship between blood lipids and the number of smoked cigarettes was evaluated.

resultsFor the purpose of this study, 7586 participants were examined. The lifetime prevalence of smoking was 19.9%, and 11.8% were current smokers. The prevalence of dyslipidemia in current smokers (54.9%) was higher than former smokers (43.9%) and in turn former smokers higher than non-smokers (38.0%). Current smokers had greater risk of abnormal HDL cholesterol [OR (95% CI), 2.28(1.98 -2.62)] and triglyceride [OR (95% CI), 1.37(1.15 -1.67)] compared to non-smokers. There was no significant difference in total cholesterol and LDL cholesterol between the two groups. A dose-response relationship was found between the number of cigarettes smoked and HDL-C and TG but no relationship was observed in terms of total cholesterol and LDL-C.

conclusionsAs compared to non-smokers, current smokers and former smokers had abnormal HDL-C and triglyceride and abnormal total cholesterol and triglyceride, respectively. After quitting smoking, heavy smokers showed a more normal HDL-C and total cholesterol levels than the people who tended to smoke a lower number of cigarettes per day.

Indexed as

AdultAgedCholesterol, HDLCholesterol, LDLCohort StudiesConfounding Factors, EpidemiologicCross-Sectional StudiesDyslipidemiasFemaleHumansIranLipidsLogistic ModelsMaleMiddle AgedPrevalenceCholesterol, HDLCholesterol, LDLLipidsTriglyceridesBlood lipidsCurrent smokerDyslipidemiaFormer smokerSmoking

Identifiers

PMID32404075
PMCPMC7218585
OpenAlexW3027421998

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.