Computational Study to Identify the Effects of the KCNJ2 E299V Mutation in Cardiac Pumping Capacity.

Open access · hybridAbstract read

In one paragraph

Article in Computational and mathematical methods in medicine, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it

0cells of the map it votes in

2citing papers in PubMed

0.7field-weighted citation impact, top 27% of its field

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 7 citations in OpenAlex.

Disruption of a Conservative Motif in the C-Terminal Loop of the KCNQ1 Channel Causes LQT Syndrome.International journal of molecular sciences · 2022
Article
A novel complex genomic rearrangement affecting the KCNJ2 regulatory region causes a variant of Cooks syndrome.Human genetics · 2022
Article

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Da Un JeongDepartment of IT Convergence Engineering, Kumoh National Institute of Technology, Gumi 39177, Republic of Korea.ORCID https://orcid.org/0000-0001-8490-5007

Jiyeong LeeDepartment of IT Convergence Engineering, Kumoh National Institute of Technology, Gumi 39177, Republic of Korea.ORCID https://orcid.org/0000-0001-7338-9756

Ki Moo LimDepartment of Medical IT Convergence Engineering, Kumoh National Institute of Technology, Gumi 39177, Republic of Korea.ORCID https://orcid.org/0000-0001-6729-8129

Kumoh National Institute of Technology · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The KCNJ2 gene mutations induce short QT syndrome (SQT3) by directly increasing the

Indexed as

Models, CardiovascularMutationArrhythmias, CardiacBiomechanical PhenomenaComputational BiologyComputer SimulationElectrophysiological PhenomenaFinite Element AnalysisHeart VentriclesHumansMyocardial ContractionPotassium Channels, Inwardly RectifyingKCNJ2 protein, humanPotassium Channels, Inwardly Rectifying

Identifiers

PMID32328155

DOI10.1155/2020/7194275

PMCPMC7150720

OpenAlexW3015179647

What OpenQuestion holds

Textmetadata

LicenceCC BY

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.