Evidence map›Paper›PMID 32273889›Full record

ReviewNeural plasticity2020

Canonical Transient Receptor Potential (TRPC) Channels in Nociception and Pathological Pain.

Zhi-Chuan Sun, Sui-Bin Ma, Wen-Guang Chu, Dong Jia, Ceng Luo

Abstract readReview
In one paragraph

Review in Neural plasticity, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. TRPC4 Mediates Trigeminal Neuropathic Pain via CaCNS neuroscience & therapeutics · 2025
    Article
  5. Review
  6. Dysregulation of sphingolipid metabolism in pain.Frontiers in pharmacology · 2024
    Review
  7. Review
  8. Review
  9. Review
  10. Article
  11. Review
  12. Review
  13. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Zhi-Chuan SunDepartment of Neurosurgery, Tangdu Hospital, Fourth Military Medical University, Xi'an 710038, China.
Sui-Bin MaDepartment of Neurobiology, School of Basic Medicine, Fourth Military Medical University, Xi'an 710032, China.
Wen-Guang ChuDepartment of Neurobiology, School of Basic Medicine, Fourth Military Medical University, Xi'an 710032, China.
Dong JiaDepartment of Neurosurgery, Tangdu Hospital, Fourth Military Medical University, Xi'an 710038, China.
Ceng LuoDepartment of Neurobiology, School of Basic Medicine, Fourth Military Medical University, Xi'an 710032, China.ORCID 0000-0003-3742-7713

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chronic pathological pain is one of the most intractable clinical problems faced by clinicians and can be devastating for patients. Despite much progress we have made in understanding chronic pain in the last decades, its underlying mechanisms remain elusive. It is assumed that abnormal increase of calcium levels in the cells is a key determinant in the transition from acute to chronic pain. Exploring molecular players mediating Ca

Indexed as

AnimalsBrainCalcium SignalingGanglia, SpinalHumansNeural PathwaysNeuronsNociceptionPainTransient Receptor Potential ChannelsTransient Receptor Potential Channels

Identifiers

PMID32273889
PMCPMC7115173

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.