Evidence map›Paper›PMID 32231670›Full record

ArticleFrontiers in immunology2020

Distinct Dysfunctional States of Circulating Innate-Like T Cells in Metabolic Disease.

Yanyan Li, Katherine Woods, Amber Parry-Strong, Regan J Anderson, Celina Capistrano, Aurelie Gestin, Gavin F Painter, Ian F Hermans, Jeremy Krebs, Olivier Gasser

Open access · goldAbstract read
In one paragraph

Article in Frontiers in immunology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.6field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Review
  5. Review
  6. Article
  7. Involvement of impaired CD8Reproductive biology and endocrinology : RB&E · 2021
    Article
  8. Article
  9. Obesity and immune status in children.Current opinion in pediatrics · 2020
    Review
  10. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 1 country.

Yanyan LiMalaghan Institute of Medical Research, Wellington, New Zealand.
Katherine WoodsMalaghan Institute of Medical Research, Wellington, New Zealand.
Amber Parry-StrongSchool of Medicine, University of Otago, Wellington, New Zealand.
Regan J AndersonFerrier Institute, Victoria University of Wellington, Wellington, New Zealand.
Celina CapistranoMalaghan Institute of Medical Research, Wellington, New Zealand.
Aurelie GestinMalaghan Institute of Medical Research, Wellington, New Zealand.
Gavin F PainterFerrier Institute, Victoria University of Wellington, Wellington, New Zealand.
Ian F HermansMalaghan Institute of Medical Research, Wellington, New Zealand.
Jeremy KrebsSchool of Medicine, University of Otago, Wellington, New Zealand.
Olivier GasserMalaghan Institute of Medical Research, Wellington, New Zealand.
Malaghan Institute of Medical Research · NZUniversity of Otago · NZVictoria University of Wellington · NZ

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The immune system plays a significant role in controlling systemic metabolism. Innate-like T (ILT) cells in particular, such as mucosal-associated invariant T (MAIT) cells, invariant natural killer T (iNKT) cells and γδ T cell receptor expressing cells, have been reported to promote metabolic homeostasis. However, these different ILT cell subsets have, to date, been generally studied in isolation. Here we conducted a pilot study assessing the phenotype and function of circulating MAIT, iNKT, and Vδ2

Indexed as

AdultAgedBlood CirculationCells, CulturedDiabetes Mellitus, Type 2FemaleHumansImmunity, InnateLymphocyte ActivationMaleMetabolic DiseasesMiddle AgedMucosal-Associated Invariant T CellsNatural Killer T-CellsObesityReceptors, Antigen, T-Cell, gamma-deltaReceptors, Antigen, T-Cell, gamma-deltaT-cell receptor Vdelta2, humaniNKT cellsMAIT cellsobesitytype 2 diabetesVδ2+ T cells

Identifiers

PMID32231670
PMCPMC7082397
OpenAlexW3011002958

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.