Evidence map›Paper›PMID 32205316›Full record

ArticleG3 (Bethesda, Md.)2020

Tools for the Genetic Manipulation of

Megan A Sloan, Petros Ligoxygakis

Open access · goldAbstract read
In one paragraph

Article in G3 (Bethesda, Md.), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.1field-weighted citation impact, top 51% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Megan A SloanDepartment of Biochemistry, University of Oxford, South Parks Rd, Oxford, OX1 3QU, UK.ORCID 0000-0001-7659-4069
Petros LigoxygakisDepartment of Biochemistry, University of Oxford, South Parks Rd, Oxford, OX1 3QU, UK petros.ligoxygakis@bioch.ox.ac.uk.ORCID 0000-0002-9498-9993
University of Oxford · GB

Funding

Biotechnology and Biological Sciences Research Council BB/K003569Wellcome Trust
6 · The paper itself

Abstract

Trypanosomatid parasites are causative agents of important human and animal diseases such as sleeping sickness and leishmaniasis. Most trypanosomatids are transmitted to their mammalian hosts by insects, often belonging to Diptera (or true flies). With resistance to both vector-targeted pesticides and trypanocidal drugs being reported, there is a need for novel transmission blocking strategies to be developed. Studies using the blood-feeding vectors themselves are not broadly accessible, as such, new model systems are being developed to unpick insect-trypanosmatids interactions. One such case is the interactions between the model dipteran

Indexed as

Drosophila melanogasterTrypanosomatinaAnimalsDrosophilaInsectaDrosophilaHerpetomonasMonoxenousParasiteTrypanosomatid

Identifiers

PMID32205316
PMCPMC7202032
OpenAlexW3012871314

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.