Evidence map›Paper›PMID 32196486›Full record

ArticlePLoS genetics2020

GLP-1 Notch-LAG-1 CSL control of the germline stem cell fate is mediated by transcriptional targets lst-1 and sygl-1.

Jian Chen, Ariz Mohammad, Nanette Pazdernik, Huiyan Huang, Beth Bowman, Eric Tycksen, Tim Schedl

Open access · goldAbstract read
In one paragraph

Article in PLoS genetics, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed
3.7field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 54 citations in OpenAlex.

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  15. LST-1 is a bifunctional regulator that feeds back on Notch-dependent transcription to regulateProceedings of the National Academy of Sciences of the United States of America · 2023
    Article
  16. Article
  17. Oogenesis in Caenorhabditis elegans.Sexual development : genetics, molecular biology, evolution, endocrinology, embryology, and pathology of sex determination and differentiation · 2023
    Review
  18. Analysis of the C. elegans Germline Stem Cell Pool.Methods in molecular biology (Clifton, N.J.) · 2023
    Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 3 institutions in 1 country.

Jian ChenDepartment of Genetics, Washington University School of Medicine, Saint Louis, Missouri, United States of America.
Ariz MohammadDepartment of Genetics, Washington University School of Medicine, Saint Louis, Missouri, United States of America.ORCID 0000-0002-2807-412X
Nanette PazdernikDepartment of Genetics, Washington University School of Medicine, Saint Louis, Missouri, United States of America.ORCID 0000-0002-1037-6437
Huiyan HuangDepartment of Pediatrics, Washington University School of Medicine, Saint Louis, Missouri, United States of America.ORCID 0000-0003-1239-7191
Beth BowmanDepartment of Biology, Emory University, Atlanta, Georgia, United States of America.ORCID 0000-0003-3733-4920
Eric TycksenGenome Technology Access Center, McDonnell Genome Institute, Washington University School of Medicine, Saint Louis, Missouri, United States of America.ORCID 0000-0001-6362-0141
Tim SchedlDepartment of Genetics, Washington University School of Medicine, Saint Louis, Missouri, United States of America.ORCID 0000-0003-2148-2996
Washington University in St. Louis · USEmory University · USJames S. McDonnell Foundation · US

Funding

Stem cell versus meiotic fate decision in C. elegansR01GM100756 · NIGMS · WASHINGTON UNIVERSITY · PI SCHEDL, TIM · 2012 to 2023
$4.2M
NIGMS NIH HHS R01 GM100756
6 · The paper itself

Abstract

Stem cell systems are essential for the development and maintenance of polarized tissues. Intercellular signaling pathways control stem cell systems, where niche cells signal stem cells to maintain the stem cell fate/self-renewal and inhibit differentiation. In the C. elegans germline, GLP-1 Notch signaling specifies the stem cell fate, employing the sequence-specific DNA binding protein LAG-1 to implement the transcriptional response. We undertook a comprehensive genome-wide approach to identify transcriptional targets of GLP-1 signaling. We expected primary response target genes to be evident at the intersection of genes identified as directly bound by LAG-1, from ChIP-seq experiments, with genes identified as requiring GLP-1 signaling for RNA accumulation, from RNA-seq analysis. Furthermore, we performed a time-course transcriptomics analysis following auxin inducible degradation of LAG-1 to distinguish between genes whose RNA level was a primary or secondary response of GLP-1 signaling. Surprisingly, only lst-1 and sygl-1, the two known target genes of GLP-1 in the germline, fulfilled these criteria, indicating that these two genes are the primary response targets of GLP-1 Notch and may be the sole germline GLP-1 signaling protein-coding transcriptional targets for mediating the stem cell fate. In addition, three secondary response genes were identified based on their timing following loss of LAG-1, their lack of a LAG-1 ChIP-seq peak and that their glp-1 dependent mRNA accumulation could be explained by a requirement for lst-1 and sygl-1 activity. Moreover, our analysis also suggests that the function of the primary response genes lst-1 and sygl-1 can account for the glp-1 dependent peak protein accumulation of FBF-2, which promotes the stem cell fate and, in part, for the spatial restriction of elevated LAG-1 accumulation to the stem cell region.

Indexed as

Adult Germline Stem CellsAnimalsCaenorhabditis elegansCaenorhabditis elegans ProteinsCell DifferentiationCell LineageDNA-Binding ProteinsGerm CellsGlucagon-Like Peptide 1Receptors, NotchRNARNA, MessengerSignal TransductionStem CellsTranscription FactorsCaenorhabditis elegans ProteinsDNA-Binding ProteinsGlp-1 protein, C elegansGlucagon-Like Peptide 1LAG-1 protein, C elegansLST-1 protein, C elegansReceptors, NotchRNARNA, MessengerSYGL-1 protein, C elegansTranscription Factors

Identifiers

PMID32196486
PMCPMC7153901
OpenAlexW3012129213

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.