Evidence map›Paper›PMID 32193953›Full record

ReviewCell transplantation

Endothelial Progenitor Cells in Moyamoya Disease: Current Situation and Controversial Issues.

Jin Yu, Qian Du, Miao Hu, Jianjian Zhang, Jincao Chen

Open access · goldAbstract readReview
In one paragraph

Review in Cell transplantation. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.4field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 14 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Review
  5. Article
  6. Article
  7. Article
  8. Article
  9. Review
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Jin YuDepartment of Neurosurgery, Zhongnan Hospital of Wuhan University, Wuhan, China.
Qian DuDepartment of Rheumatology, Xiangya Hospital, Central South University, Changsha, Hunan, China.
Miao HuDepartment of Neurosurgery, Zhongnan Hospital of Wuhan University, Wuhan, China.
Jianjian ZhangDepartment of Neurosurgery, Zhongnan Hospital of Wuhan University, Wuhan, China.ORCID 0000-0001-9882-7978
Jincao ChenDepartment of Neurosurgery, Zhongnan Hospital of Wuhan University, Wuhan, China.ORCID 0000-0002-9071-9670
Wuhan University · CNCentral South University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Due to the lack of animal models and difficulty in obtaining specimens, the study of pathogenesis of moyamoya disease (MMD) almost stagnated. In recent years, endothelial progenitor cells (EPCs) have attracted more and more attention in vascular diseases due to their important role in neovascularization. With the aid of paradigms and methods in cardiovascular diseases research, people began to explore the role of EPCs in the processing of MMD. In the past decade, studies have shown that abnormalities in cell amounts and functions of EPCs were closely related to the vascular pathological changes in MMD. However, the lack of consistent criteria, such as isolation, cultivation, and identification standards, is also blocking the way forward. The goal of this review is to provide an overview of the current situation and controversial issues relevant to studies about EPCs in the pathogenesis and etiology of MMD.

Indexed as

AnimalsCell DifferentiationDisease Models, AnimalDisease ProgressionEndothelial Progenitor CellsHumansMoyamoya DiseaseNeovascularization, Pathologicendothelial progenitor cellsmoyamoya diseaseneovascularizationpathogenesis

Identifiers

PMID32193953
PMCPMC7444216
OpenAlexW3011464205

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.