ArticleProceedings. Biological sciences2020
Prevalence, infected density or individual probability of infection? Assessing vector infection risk in the wild transmission of Chagas disease.
Article in Proceedings. Biological sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
9 citing papers in PubMed.
- Empowering communities through One Health and ArtScience: An innovative approach to combat Chagas disease in endemic areas of Minas Gerais, Brazil.Public health challenges · 2025Article
- Humans as blood-feeding sources in sylvatic triatomines of Chile unveiled by next-generation sequencing.Parasites & vectors · 2023Article
- NaturalInsects · 2023Article
- Estimating the genetic structure of Triatoma dimidiata (Hemiptera: Reduviidae) and the transmission dynamics of Trypanosoma cruzi in Boyacá, eastern Colombia.PLoS neglected tropical diseases · 2022Article
- Testing Phylogeographic Hypotheses inInsects · 2022Article
- Blood-Meal Sources andMicroorganisms · 2022Article
- Over-dispersed Trypanosoma cruzi parasite load in sylvatic and domestic mammals and humans from northeastern Argentina.Parasites & vectors · 2022Article
- Trypanosoma cruzi infection follow-up in a sylvatic vector of Chagas disease: Comparing early and late stage nymphs.PLoS neglected tropical diseases · 2021Article
- Trypanosomatid Infections among Vertebrates of Chile: A Systematic Review.Pathogens (Basel, Switzerland) · 2020Review
Corrections and comments
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Authors and funding
6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Vector-borne infectious disease dynamics result mainly from the intertwined effect of the diversity, abundance, and behaviour of hosts and vectors. Most studies, however, have analysed the relationship between host-species diversity and infection risk, focusing on vector population instead of individuals, probably dismissing the level at which the transmission process occurs. In this paper, we examine the importance of the host community in accounting for infection risk, at both population and individual levels, using the wild transmission of the protozoan that causes Chagas disease as a vector-borne disease model. Chagas disease is caused by
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Registered trials
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