Evidence map›Paper›PMID 32148911›Full record

ArticleInterventional medicine & applied science2019

Evaluation of homocystein and asymmetric dimethyl arginine levels in patients with coronary slow flow phenomenon.

Erkan Demirci, Oğuzhan Çelik, Macit Kalçık, Lütfü Bekar, Mucahit Yetim, Tolga Doğan

Open access · diamondAbstract read
In one paragraph

Article in Interventional medicine & applied science, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed, 1 pooled it
1.5field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 1 synthesis or guideline pooled it, 13 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Article
  4. Article
  5. Article
  6. Coronary slow flow research: a bibliometric analysis.European journal of medical research · 2023
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Erkan DemirciDepartment of Cardiology, Kayseri Training and Research Hospital, Kayseri, Turkey.
Oğuzhan ÇelikDepartment of Cardiology, Muğla Sıtkı Koçman University Training and Research Hospital, Muğla, Turkey.
Macit KalçıkDepartment of Cardiology, Faculty of Medicine, Hitit University, Çorum, Turkey.
Lütfü BekarDepartment of Cardiology, Faculty of Medicine, Hitit University, Çorum, Turkey.
Mucahit YetimDepartment of Cardiology, Faculty of Medicine, Hitit University, Çorum, Turkey.
Tolga DoğanDepartment of Cardiology, Faculty of Medicine, Hitit University, Çorum, Turkey.
Hitit Üniversitesi · TRKayseri Eğitim ve Araştırma Hastanesi · TRMuğla University · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundPrevious studies have demonstrated that homocysteine and asymmetric dimethyl arginine (ADMA) levels were strongly associated with cardiovascular diseases including coronary artery disease. The aim of this study was to investigate the role of plasma homocysteine and ADMA levels in the pathogenesis of coronary slow flow (CSF) phenomenon.

methodsTwenty-three patients with CSF and 25 controls with normal coronary flow were included in this study. The quantitative measurement of coronary blood flow was performed using the thrombolysis in myocardial infarction frame count method. Plasma homocysteine and ADMA levels were determined using enzymatic assays from venous blood samples.

resultsThe patients with CSF had significantly higher plasma homocysteine levels than controls (16.2 ± 7.6 vs. 12.2 ± 2.2 μM/L;

conclusionsIncreased homocysteine and uric acid levels may play an important role in the pathogenesis of CSF. Further large scale studies are required to determine the relationship between ADMA levels and CSF.

Indexed as

asymmetric dimethyl argininecoronary arterycoronary slow flowendothelial dysfunctionhomocystein

Identifiers

PMID32148911
PMCPMC7044542
OpenAlexW2945322290

What OpenQuestion holds

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LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.