Evidence map›Paper›PMID 32145019›Full record

ArticleGenome biology and evolution2020

Genome Sequence of an Emerging Salmonella enterica Serovar Infantis and Genomic Comparison with Other S. Infantis Strains.

Emiliano Cohen, Galia Rahav, Ohad Gal-Mor

Abstract read
In one paragraph

Article in Genome biology and evolution, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed.

  1. Article
  2. Novel pESI-encoded autotransporter adhesin PeaP of epidemicbioRxiv : the preprint server for biology · 2026
    Article
  3. Genomic Evidence of Multidrug-ResistantbioRxiv : the preprint server for biology · 2026
    Article
  4. Phenotypic Expression ofMicroorganisms · 2026
    Article
  5. Prophage diversity in poultry-associatedFrontiers in microbiology · 2026
    Article
  6. A rare case of gastroenteritis due toFrontiers in public health · 2026
    Article
  7. Epidemiology and antimicrobial resistance ofInfectious diseases (London, England) · 2026
    Article
  8. Article
  9. Antibiotic-ResistantFood science of animal resources · 2025
    Review
  10. Mutation inVeterinary sciences · 2024
    Article
  11. Article
  12. Article
  13. Effects ofMicroorganisms · 2023
    Article
  14. Article
  15. Article
  16. Article
  17. Genomic Analysis of Two MDR Isolates ofAntibiotics (Basel, Switzerland) · 2022
    Article
  18. Article
  19. What makes a megaplasmid?Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2022
    Article
  20. Evaluation of Antibiotic Resistance ofAntibiotics (Basel, Switzerland) · 2021
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Emiliano CohenThe Infectious Diseases Research Laboratory, Sheba Medical Center, Tel-Hashomer, Israel.
Galia RahavThe Infectious Diseases Research Laboratory, Sheba Medical Center, Tel-Hashomer, Israel.
Ohad Gal-MorThe Infectious Diseases Research Laboratory, Sheba Medical Center, Tel-Hashomer, Israel.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Salmonella enterica serovar Infantis (S. Infantis) is one of the dominant serovars of the bacterial pathogen S. enterica. In recent years, the number of human infections caused by S. Infantis has been increasing in many countries, and often the emerging population harbors a unique virulence-resistant megaplasmid called plasmid of emerging S. Infantis (pESI). Here, we report the complete gap-free genome sequence of the S. Infantis Israeli emerging clone and compare its chromosome and pESI sequences with other complete S. Infantis genomes. We show a conserved presence of the Salmonella pathogenicity islands 1-6, 9, 11, 12, and CS54 and a common integration of five bacteriophages in the S. Infantis chromosome. In contrast, we found variable presence of additionally three chromosomally integrated phages and eight modular regions in pESI, which contribute to the genetic and phenotypic diversity (including antimicrobial resistance) of this ubiquitous foodborne pathogen.

Indexed as

Genome, BacterialGenomic IslandsGenomicsPlasmidsSalmonella entericaVirulenceHGTmobile genetic elementspESISalmonella InfantisvirulenceWGS

Identifiers

PMID32145019
PMCPMC7144548

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.