Evidence map›Paper›PMID 32082426›Full record

ArticleMobile DNA2020

Hybrid dysgenesis in

Lucas W Hemmer, Guilherme B Dias, Brittny Smith, Kelley Van Vaerenberghe, Ashley Howard, Casey M Bergman, Justin P Blumenstiel

Open access · goldAbstract read
In one paragraph

Article in Mobile DNA, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
3.0field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 21 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Paramutation-Like Behavior of Genic piRNA-Producing Loci inInternational journal of molecular sciences · 2025
    Article
  6. Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Review
  12. Review
  13. Article
  14. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Lucas W Hemmer1Department of Ecology and Evolutionary Biology, University of Kansas, Lawrence, KS 66045 USA.ORCID 0000-0002-8036-5207
Guilherme B Dias3Department of Genetics and Institute of Bioinformatics, University of Georgia, Athens, GA 30602 USA.
Brittny Smith4Department of Molecular Biosciences, University of Kansas, Lawrence, KS 66045 USA.
Kelley Van Vaerenberghe1Department of Ecology and Evolutionary Biology, University of Kansas, Lawrence, KS 66045 USA.
Ashley Howard1Department of Ecology and Evolutionary Biology, University of Kansas, Lawrence, KS 66045 USA.
Casey M Bergman3Department of Genetics and Institute of Bioinformatics, University of Georgia, Athens, GA 30602 USA.
Justin P Blumenstiel1Department of Ecology and Evolutionary Biology, University of Kansas, Lawrence, KS 66045 USA.
University of Kansas · USUniversity of Georgia · US

Funding

Understanding the mechanobiology of stem cells in a microengineered 3D cardiac tissue environment with cardiomyopathyP20GM103638 · NIGMS · UNIVERSITY OF KANSAS LAWRENCE · PI LUNTE, SUSAN M · 2012 to 2021
$22.1M
A resource for the genetic analysis of complex traitsR01OD010974 · OD · UNIVERSITY OF KANSAS LAWRENCE · PI LONG, ANTHONY DOUGLAS, MACDONALD, STUART JOHN · 2012 to 2019
$3.1M
NIGMS NIH HHS P20 GM103638NIH HHS R01 OD010974
6 · The paper itself

Abstract

backgroundTransposable elements (TEs) are endogenous mutagens and their harmful effects are especially evident in syndromes of hybrid dysgenesis. In

resultsUsing the

conclusionsOverall, we conclude that increased mitotic recombination is likely the result of early TE activation in dysgenic progeny, but a stable landscape of meiotic recombination indicates that either transposition is ameliorated in the adult female germline or that regulation of meiotic recombination is robust to ongoing transposition. These results indicate that the effects of parental TE asymmetry on recombination are likely sensitive to the timing of transposition.

Indexed as

Drosophila virilisHybrid dysgenesisMeiotic recombinationMitotic recombinationTransposons

Identifiers

PMID32082426
PMCPMC7023781
OpenAlexW3011490953

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.