Evidence map›Paper›PMID 32078066›Full record

ReviewCurrent atherosclerosis reports2020

Analyzing Preeclampsia as the Tip of the Iceberg Represented by Women with Long-Term Cardiovascular Disease, Atherosclerosis, and Inflammation.

Angélica Lemos Debs Diniz, Maria Marta Bini Martins Paes, Aline Debs Diniz

Abstract readReview
PubMed Publisher
In one paragraph

Review in Current atherosclerosis reports, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed, 1 pooled it
1.6field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed, 1 synthesis or guideline pooled it, 21 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Metabolic theory of preeclampsia: implications for maternal cardiovascular health.American journal of physiology. Heart and circulatory physiology · 2024
    Review
  4. Article
  5. Article
  6. Article
  7. Inorganic Phosphate in the Pathogenesis of Pregnancy-Related Complications.International journal of molecular sciences · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Angélica Lemos Debs DinizDepartment of Gynecology and Obstetrics, Federal University of Uberlândia (UFU), Avenida Pará SN, Campus Umuarama, Uberlândia, Minas Gerais, 38400-902, Brazil. angelicadiniz@ufu.br.
Maria Marta Bini Martins PaesClinical Hospital, Federal University of Uberlândia - UFU, Avenida Pará SN, Campus Umuarama, Uberlândia, Minas Gerais, 38400-902, Brazil.
Aline Debs DinizPositive University, R. Prof. Pedro Viriato Parigot de Souza, 5300 - Cidade Industrial, Curitiba, Paraná, 81280-330, Brazil.
Universidade Federal de Uberlândia · BRUniversidade Positivo · BR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

purpose of reviewCardiovascular and endothelial dysfunction is recognized nowadays as an important etiological factor contributing to the development of hypertensive disorders of pregnancy. RECENT

findingsPreeclampsia is considered a specific disease of pregnancy, but recent theories suggest that women suffering from the condition have greater propensity to develop atherosclerosis, heart disease, and stroke over the years. It is possible that transient but severe endothelial dysfunction observed in preeclampsia potentiates a cascade of events that progresses to atherosclerosis. Preeclampsia offers a unique window of opportunity to identify maternal endothelial dysfunction and pre-existing cardiovascular disease. The placenta is closely involved in the onset of preeclampsia, but endothelial and cardiac vascular factors also play important causal roles in the development of hypertension during pregnancy. According to the data presented, it is clear that preeclampsia selects a group at high risk of development of atherosclerosis and at increased cardiovascular risk, as well as of stroke, in the decades following childbirth.

Indexed as

AspirinAtherosclerosisFemaleHumansHypertensionInflammationPlacentaPlacentationPre-EclampsiaPregnancyRisk FactorsAspirinArterial stiffnessAtherosclerosisCardiovascular riskDopplerEclampsiaEndothelial dysfunctionGestationHypertensive disordersInflammationOphthalmic arteryPreeclampsia

Identifiers

PMID32078066
OpenAlexW3008069053

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.