Evidence map›Paper›PMID 32075856›Full record

ArticleJournal of lipid research2020

Hexacosenoyl-CoA is the most abundant very long-chain acyl-CoA in ATP binding cassette transporter D1-deficient cells.

Kotaro Hama, Yuko Fujiwara, Shigeo Takashima, Yasuhiro Hayashi, Atsushi Yamashita, Nobuyuki Shimozawa, Kazuaki Yokoyama

Open access · hybridAbstract read
In one paragraph

Article in Journal of lipid research, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.8field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 18 citations in OpenAlex.

  1. ACAD10 and ACAD11 enable mammalian 4-hydroxy acid lipid catabolism.Nature structural & molecular biology · 2025
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Kotaro HamaFaculty of Pharma-Science, Teikyo University, Itabashi-ku, Tokyo 173-8605, Japan khama@pharm.teikyo-u.ac.jp.
Yuko FujiwaraFaculty of Pharma-Science, Teikyo University, Itabashi-ku, Tokyo 173-8605, Japan.
Shigeo TakashimaDivision of Genomics Research, Life Science Research Center, Gifu University, Gifu 501-1193, Japan.ORCID 0000-0002-2273-1643
Yasuhiro HayashiFaculty of Pharma-Science, Teikyo University, Itabashi-ku, Tokyo 173-8605, Japan.
Atsushi YamashitaFaculty of Pharma-Science, Teikyo University, Itabashi-ku, Tokyo 173-8605, Japan.ORCID 0000-0002-2544-2134
Nobuyuki ShimozawaDivision of Genomics Research, Life Science Research Center, Gifu University, Gifu 501-1193, Japan.
Kazuaki YokoyamaFaculty of Pharma-Science, Teikyo University, Itabashi-ku, Tokyo 173-8605, Japan.
Teikyo University · JPGifu University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

X-linked adrenoleukodystrophy (X-ALD) is an inherited disorder caused by deleterious mutations in the

Indexed as

Acyl Coenzyme AATP Binding Cassette Transporter, Subfamily D, Member 1FibroblastsGene Knockout TechniquesHeLa CellsHumansAcyl Coenzyme AATP Binding Cassette Transporter, Subfamily D, Member 1adenosine 5′-triphosphatefatty acidfatty acid/metabolismhexacosenoyl (26:1)-coenzyme Ainherited metabolic disorderlipidomicslipidsperoxisomesvery long-chain fatty acidX-linked adrenoleukodystrophy

Identifiers

PMID32075856
PMCPMC7112142
OpenAlexW3008249294

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.