Evidence map›Paper›PMID 32019113›Full record

ReviewInternational journal of molecular sciences2020

Genetic Dissection of Alzheimer's Disease Using

Youngjae Jeon, Jae Ha Lee, Byoungyun Choi, So-Yoon Won, Kyoung Sang Cho

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed.

  1. Article
  2. From development to disease: the neuronal role of hnRNPs through the lens ofFrontiers in cell and developmental biology · 2026
    Review
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Review
  9. Article
  10. Article
  11. Review
  12. Review
  13. Article
  14. Journal of developmental biology · 2022
    Review
  15. Review
  16. Article
  17. Review
  18. Article
  19. Pharmacological Treatment of Alzheimer's Disease: Insights fromInternational journal of molecular sciences · 2020
    Review
  20. Aβ Plaques.Free neuropathology · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Youngjae JeonDepartment of Biological Sciences, Konkuk University, Seoul 05029, Korea.ORCID 0000-0001-8206-6772
Jae Ha LeeDepartment of Biological Sciences, Konkuk University, Seoul 05029, Korea.
Byoungyun ChoiDepartment of Biological Sciences, Konkuk University, Seoul 05029, Korea.ORCID 0000-0002-6561-0446
So-Yoon WonDepartment of Biochemistry, Chungbuk National University College of Medicine, Cheongju 28644, Korea.
Kyoung Sang ChoDepartment of Biological Sciences, Konkuk University, Seoul 05029, Korea.ORCID 0000-0002-3277-8269

Funding

Korea Health Industry Development Institute HI18C0041
6 · The paper itself

Abstract

Alzheimer's disease (AD), a main cause of dementia, is the most common neurodegenerative disease that is related to abnormal accumulation of the amyloid β (Aβ) protein. Despite decades of intensive research, the mechanisms underlying AD remain elusive, and the only available treatment remains symptomatic. Molecular understanding of the pathogenesis and progression of AD is necessary to develop disease-modifying treatment.

Indexed as

Alzheimer DiseaseAmyloid beta-PeptidesAnimalsBrainDisease Models, AnimalDrosophilaGenomicsHumansNeurodegenerative DiseasesAmyloid beta-PeptidesAD modelAlzheimer’s diseaseamyloid βDrosophilafunctional genomics

Identifiers

PMID32019113
PMCPMC7037931

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.