Evidence map›Paper›PMID 31993331›Full record

ReviewToxicology reports2019

Oxidative damage and antioxidative system in algae.

Maryam Rezayian, Vahid Niknam, Hassan Ebrahimzadeh

Abstract readReview
In one paragraph

Review in Toxicology reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 90 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
90citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

90 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
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  5. Article
  6. Review
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  9. Article
  10. Review
  11. Article
  12. Physiological and Molecular Response Mechanisms ofInternational journal of molecular sciences · 2026
    Article
  13. Review
  14. Oxidative stress inFrontiers in plant science · 2026
    Review
  15. Article
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article

30 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Maryam RezayianDepartment of Plant Biology, and Center of Excellence in Phylogeny of Living Organisms in Iran, School of Biology, College of Science, University of Tehran, Tehran 14155, Iran.
Vahid NiknamDepartment of Plant Biology, and Center of Excellence in Phylogeny of Living Organisms in Iran, School of Biology, College of Science, University of Tehran, Tehran 14155, Iran.
Hassan EbrahimzadehDepartment of Plant Biology, and Center of Excellence in Phylogeny of Living Organisms in Iran, School of Biology, College of Science, University of Tehran, Tehran 14155, Iran.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Reactive oxygen species (ROS) typically produce in algae and act as secondary messengers in numerous cellular processes. Under abiotic stresses, the balance between production and suppression of ROS disappears and causes increase of ROS. Increasing excessive ROS can cause damage to various cellular components comprising cell membranes, proteins and lipids. Algae have an antioxidant defense system to overcome on oxidative damage. Antioxidant defense mechanisms are of two types, namely enzymatic and non-enzymatic antioxidants. The enzymatic antioxidants include superoxide dismutase, catalase, ascorbate peroxidase and glutathione reductase. The non-enzymatic antioxidants include carotenoids, tocopherol, ascorbic acid, glutathione, flavonoids and phenolic compounds. In this review, we describe the various types of ROS and their production, and antioxidant defense mechanisms for ROS suppression.

Indexed as

Enzymatic antioxidantsNon-enzymatic antioxidantsOxidative stressReactive oxygen species

Identifiers

PMID31993331
PMCPMC6978204

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.