ArticleJournal of cardiology cases2019
Transcoronary mapping and chemical ablation of ventricular tachycardia in no-entry left ventricle.
Article in Journal of cardiology cases, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
2 citing papers in PubMed, 7 citations in OpenAlex.
- Guidewire Ablation within the Coronary Venous System for Epicardial or Intramural Ventricular Arrhythmia: A Preclinical Study of Biophysical Characterization.Cardiology research and practice · 2024Article
- Transcoronary mapping with an over-the-wire multielectrode catheter in scar-related ventricular tachycardia patients.Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology · 2023Article
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
It is challenging to perform ablation of ventricular tachycardia (VT) from the left ventricle (LV) in patients without catheter access to the LV. A 50-year-old man was referred to our hospital for VT. He underwent mechanical aortic and mitral valve replacement for infective endocarditis and embolic myocardial infarction in the left ventricular inferior wall during a surgery. Anti-arrhythmia drugs (AADs) such as sotalol and bisoprolol were initiated and implantable cardioverter defibrillator was implanted. However, 2 months after discharge, he was admitted again for cardiac implantable electronic device (CIED) infection and underwent complete CIED system removal. During hospitalization, VT easily occurred despite the use of AADs. We decided to perform transcoronary chemical ablation to treat this drug-refractory VT. A 0.014-inch guide-wire and a micro-catheter were advanced into coronary arteries. Pace map was conducted using a guide-wire and the micro artery branch feeding the VT exit area was detected. Ethanol infusion to this branch and the slightly basal side of the area eliminated the VT. We successfully treated VT in the no-entry LV by wire-guided mapping and ethanol ablation via coronary arteries. VT has not recurred during the follow-up period of 12 months. <
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