ArticleClinical and translational allergy2020
Indoor mold odor in the workplace increases the risk of Asthma-COPD Overlap Syndrome: a population-based incident case-control study.
Article in Clinical and translational allergy, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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Who cites it
6 citing papers in PubMed, 14 citations in OpenAlex.
- Respiratory Diseases Associated With Organic Dust Exposure.The journal of allergy and clinical immunology. In practice · 2024Review
- AWMF mold guideline "Medical clinical diagnostics for indoor mold exposure" - Update 2023 AWMF Register No. 161/001.Allergologie select · 2024Article
- Insights into uncovered public health risks. The case of asthma attacks among archival workers: a cross-sectional study.Frontiers in public health · 2024Article
- What should be tested in patients with suspected mold exposure? Usefulness of serological markers for the diagnosis.Allergologie select · 2022Article
- Occupation and subcategories of asthma: a population-based incident case-control study.Occupational and environmental medicine · 2021Article
- Association of toxic indoor air with multi-organ symptoms in pupils attending a moisture-damaged school in Finland.American journal of clinical and experimental immunology · 2020Article
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Authors and funding
3 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundPrevious studies have suggested an increased risk of asthma related to indoor dampness problems, but their role in the etiology of Asthma-COPD Overlap Syndrome has not been studied. We utilized a population-based incident case-control study to assess potential effect of indoor dampness and molds at home and at work on development of ACOS.
methodsWe recruited systematically all new cases of asthma diagnosed during a 2.5-year study period (1997-2000) and randomly selected controls from the source population of adults 21-63 years old and representing 500,000 persons-years in the Pirkanmaa Hospital District, South Finland. Exposure indicators included water damage, damp stains or paint peeling, visible mold, and mold odor, asked separately for home and workplace. The clinically diagnosed case series consisted of 521 adults with newly diagnosed asthma. Altogether 25 of them satisfied the criteria for ACOS-cases, i.e. FEV1/FVC < 0.70 in post-bronchodilator spirometry. The control series, including 932 controls, were from a random sample of source population, after excluding 76 (7.5%) controls with asthma.
resultsIn logistic regression analysis adjusting for confounders, the risk of ACOS was significantly related to presence of mold odor in the workplace (OR 3.43; 95% CI 1.04-11.29), but not to other dampness indicators. The fraction of ACOS attributable to workplace mold odor was 70.8% (95% CI 3.8-91.1%) among the exposed. The risk of ACOS was not related to mold exposures at home.
conclusionsPresent results provide new evidence of the significant relation between workplace exposure to mold odor and adult-onset ACOS.
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