Evidence map›Paper›PMID 31921616›Full record

ArticleFrontiers in oncology2019

Silencing of Long Non-coding RNA SMAD5-AS1 Reverses Epithelial Mesenchymal Transition in Nasopharyngeal Carcinoma via microRNA-195-Dependent Inhibition of SMAD5.

Siwei Li, Bo Zhao, Haiying Zhao, Cui Shang, Man Zhang, Xiaoxia Xiong, Jinjin Pu, Bohua Kuang, Guangrui Deng

Open access · goldAbstract read
In one paragraph

Article in Frontiers in oncology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
0.8field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 11 citations in OpenAlex.

  1. Increased expression of SMAD5-AS1 in glioma is associated with patient prognosis and immunity.International journal of clinical and experimental pathology · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Siwei LiDepartment of Oncology, Tongji Huangzhou Hospital, Huazhong University of Science and Technology, Huanggang, China.
Bo ZhaoDepartment of Radiation Oncology, The Affiliated Hospital of Guilin Medical University, Guilin, China.
Haiying ZhaoGraduate School, Guillin Medical University, Guilin, China.
Cui ShangDepartment of Oncology, Tongji Huangzhou Hospital, Huazhong University of Science and Technology, Huanggang, China.
Man ZhangDepartment of Oncology, Tongji Huangzhou Hospital, Huazhong University of Science and Technology, Huanggang, China.
Xiaoxia XiongDepartment of Oncology, Tongji Huangzhou Hospital, Huazhong University of Science and Technology, Huanggang, China.
Jinjin PuGraduate School, Guillin Medical University, Guilin, China.
Bohua KuangCancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.
Guangrui DengDepartment of Oncology, Tongji Huangzhou Hospital, Huazhong University of Science and Technology, Huanggang, China.
Guilin Medical University · CNHuazhong University of Science and Technology · CNUnion Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Long non-coding RNAs (lncRNAs) have gained widespread attention in recent years as a key regulator of diverse biological processes, but the knowledge of the mechanisms by which they act is still very limited. Differentially expressed lncRNA SMAD5 antisense RNA 1 (SMAD5-AS1) in nasopharyngeal carcinoma (NPC) and normal samples shown by

Indexed as

apoptosisepithelial-mesenchymal transitioninvasionmicroRNA-195migrationnasopharyngeal carcinomaSMAD5SMAD5-AS1

Identifiers

PMID31921616
PMCPMC6923203
OpenAlexW3003914232

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.