Evidence map›Paper›PMID 31919190›Full record

ArticleGenes & development2020

Hap2-Ino80-facilitated transcription promotes de novo establishment of CENP-A chromatin.

Puneet P Singh, Manu Shukla, Sharon A White, Marcel Lafos, Pin Tong, Tatsiana Auchynnikava, Christos Spanos, Juri Rappsilber, Alison L Pidoux, Robin C Allshire

Open access · diamondAbstract read
In one paragraph

Article in Genes & development, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 24 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Frontiers in epigenetics and epigenomics · 2024
    Article
  5. Article
  6. An essential role for the Ino80 chromatin remodeling complex in regulation of gene expression during cellular quiescence.Chromosome research : an international journal on the molecular, supramolecular and evolutionary aspects of chromosome biology · 2023
    Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Article
  12. Review
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  14. Article
  15. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 2 countries.

Puneet P SinghWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Manu ShuklaWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Sharon A WhiteWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Marcel LafosWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Pin TongWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Tatsiana AuchynnikavaWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Christos SpanosWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Juri RappsilberWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Alison L PidouxWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
Robin C AllshireWellcome Centre for Cell Biology, School of Biological Sciences, The University of Edinburgh, Edinburgh EH9 3BF, United Kingdom.
University of Edinburgh · GBWellcome Centre for Cell Biology · GBTechnische Universität Berlin · DE

Funding

Wellcome Trust 095021Wellcome Trust 103139Wellcome Trust 103139/Z/13/ZWellcome Trust 108504Wellcome Trust 200885Wellcome Trust 200885/Z/16/ZWellcome Trust 203149
6 · The paper itself

Abstract

Centromeres are maintained epigenetically by the presence of CENP-A, an evolutionarily conserved histone H3 variant, which directs kinetochore assembly and hence centromere function. To identify factors that promote assembly of CENP-A chromatin, we affinity-selected solubilized fission yeast CENP-A

Indexed as

ChromatinChromosomal Proteins, Non-HistoneChromosomes, FungalDNA, FungalSchizosaccharomycesSchizosaccharomyces pombe ProteinsTranscription FactorsTranscription, GeneticChromatinChromosomal Proteins, Non-HistoneCnp1 protein, S pombeDNA, FungalIno80 protein, S pombeSchizosaccharomyces pombe ProteinsTranscription FactorsCENP-Acentromerechromatinchromosome segregationepigeneticfission yeasthistonekinetochoreremodelingtranscription

Identifiers

PMID31919190
PMCPMC7000912
OpenAlexW2999334675

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.