Genetic interactions between

Dieter Maier, Anja C Nagel, Anette Preiss

Open access · goldAbstract read

In one paragraph

Article in Hereditas, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it

0cells of the map it votes in

1citing papers in PubMed

0.1field-weighted citation impact, top 55% of its field

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

Nucleo-cytoplasmic shuttling of murine RBPJ by Hairless protein matches that of Su(H) protein in the model system Drosophila melanogaster.Hereditas · 2021
Article

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Dieter Maier *Universität Hohenheim, Institut für Genetik (240A), Garbenstr. 30, 70599 Stuttgart, Germany.

Anja C Nagel *Universität Hohenheim, Institut für Genetik (240A), Garbenstr. 30, 70599 Stuttgart, Germany.

Anette PreissUniversität Hohenheim, Institut für Genetik (240A), Garbenstr. 30, 70599 Stuttgart, Germany.ORCID 0000-0002-6410-1586

University of Hohenheim · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundIn

resultsWe have identified Protein Kinase D (PKD) as a strong modifier of the

conclusionsWe propose that PKD-mediated phosphorylation is involved in underlying processes causing the

Indexed as

Epistasis, GeneticMutationAnimalsDrosophila melanogasterDrosophila ProteinsEyeEye ProteinsGenetic Association StudiesOrganogenesisPhenotypeProtein Kinase CDrosophila ProteinsEye ProteinsL protein, DrosophilaProtein Kinase Cprotein kinase DDrosophila melanogasterEye developmentLobePKD null mutantProtein kinase D

Identifiers

PMID31889943

DOI10.1186/s41065-019-0113-9

PMCPMC6924039

OpenAlexW2994905533

What OpenQuestion holds

Textmetadata

LicenceCC BY

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.