ArticleESC heart failure2020
Dilated cardiomyopathy and aldosteronoma: a causal link?
Article in ESC heart failure, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
3 citing papers in PubMed, 5 citations in OpenAlex.
- Tyrosine phosphatase Shp2 accelerated the progression of dilated cardiomyopathy via upregulating NF-κB/Src/FAK signaling induced ROS and mitophagy.Scientific reports · 2025Article
- Reversible dilated cardiomyopathy caused by primary hyperparathyroidism: A case report.World journal of cardiology · 2025Article
- Dilated cardiomyopathy and aldosteronoma: a causal link?ESC heart failure · 2020Article
Corrections and comments
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Authors and funding
3 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The aim of this study is to reveal the causal relationship between dilated cardiomyopathy and aldosteronoma. A 44-year-old male dilated cardiomyopathy patient with aldosteronoma, who demonstrated a worse cardiac function after 1 year therapy with optimized dosage of sacubirtil/valsartan, furosemide, metoprolol, and spironolactone. The patient shows a promising prognosis after aldosteronoma removal procedure. Aldosteronoma may cause dilated cardiomyopathy. We assume that the optimal treatment for aldosteronoma-induced dilated cardiomyopathy is surgical removal combined with drugs.
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Registered trials
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