Evidence map›Paper›PMID 31842455›Full record

ArticleInternational journal of molecular sciences2019

The Mechanism of Bisphenol A Atherogenicity Involves Apolipoprotein A-I Downregulation through NF-κB Activation.

Violeta G Trusca, Madalina Dumitrescu, Ioana M Fenyo, Irina F Tudorache, Maya Simionescu, Anca V Gafencu

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.9field-weighted citation impact, top 28% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Review
  3. Food science & nutrition · 2023
    Article
  4. Review
  5. Review
  6. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Violeta G TruscaInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.
Madalina DumitrescuInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.
Ioana M FenyoInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.
Irina F TudoracheInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.
Maya SimionescuInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.
Anca V GafencuInstitute of Cellular Biology and Pathology "Nicolae Simionescu", 050568 Bucharest, Romania.

Funding

European Regional Development Fund through the Competitiveness Operational Program 2014-2020 POC-A.1-A.1.1.4-E-2015, ID: P_37_668Romanian Academy RoUEFISCDI - Romania PN-III-P1-1.1-PD-2016UEFISCDI - Romania PN-III-P1-1.2-PCCDI-2017-0697
6 · The paper itself

Abstract

Apolipoprotein A-I (apoA-I) is the major protein component of high-density lipoproteins (HDL), mediating many of its atheroprotective properties. Increasing data reveal the pro-atherogenic effects of bisphenol A (BPA), one of the most prevalent environmental chemicals. In this study, we investigated the mechanisms by which BPA exerts pro-atherogenic effects. For this, LDLR

Indexed as

Gene Expression RegulationAir Pollutants, OccupationalAnimalsApolipoprotein A-IAtherosclerosisBenzhydryl CompoundsBiomarkersBisphenol A CompoundsDisease Models, AnimalFemaleHepatocytesLipidsMaleMiceMice, KnockoutNF-kappa BAir Pollutants, OccupationalApolipoprotein A-IBenzhydryl CompoundsBiomarkersbisphenol ABisphenol A CompoundsLipidsNF-kappa BPhenolsReceptors, LDLapoA-Iapolipoproteinbisphenol AhepatocytesNF-κBpromoter

Identifiers

PMID31842455
PMCPMC6941038
OpenAlexW2995053686

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.