Evidence map›Paper›PMID 31824648›Full record

ReviewF1000Research2019

Understanding diverse TRPV1 signaling - an update.

Michael C. Andresen

Abstract readReview
In one paragraph

Review in F1000Research, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. A novel role for TRPV1 in macrophage giant cell formation.bioRxiv : the preprint server for biology · 2026
    Article
  2. Review
  3. Article
  4. Article
  5. Article
  6. Article
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Michael C. AndresenDepartment of Physiology and Pharmacology, Oregon Health & Science University, Portland, OR, 97239, USA.ORCID 0000-0003-0524-1172

Funding

Lipid signaling in cardiovascular afferent transmissionR01HL133505 · NHLBI · OREGON HEALTH & SCIENCE UNIVERSITY · PI ANDRESEN, MICHAEL CHRISTIAN · 2016 to 2019
$1.8M
NHLBI NIH HHS R01 HL133505
6 · The paper itself

Abstract

The transient receptor potential vanilloid 1 (TRPV1) is densely expressed in spinal sensory neurons as well as in cranial sensory neurons, including their central terminal endings. Recent work in the less familiar cranial sensory neurons, despite their many similarities with spinal sensory neurons, suggest that TRPV1 acts as a calcium channel to release a discrete population of synaptic vesicles. The modular and independent regulation of release offers new questions about nanodomain organization of release and selective actions of G protein-coupled receptors.

Indexed as

Signal TransductionAnimalsExcitatory Postsynaptic PotentialsNeurons, AfferentSolitary NucleusTRPV Cation ChannelsTRPV Cation Channelscranial nerveNTSsolitary tract nucleussynaptic transmissionTRPV1vagusvisceral afferent

Identifiers

PMID31824648
PMCPMC6880261

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.