Evidence map›Paper›PMID 31824568›Full record

ArticleFrontiers in genetics2019

Dissecting the Role of NF-κb Protein Family and Its Regulators in Rheumatoid Arthritis Using Weighted Gene Co-Expression Network.

Jamal S M Sabir, Abdelfatteh El Omri, Babajan Banaganapalli, Majed A Al-Shaeri, Naser A Alkenani, Mumdooh J Sabir, Nahid H Hajrah, Houda Zrelli, Lukasz Ciesla, Khalidah K Nasser and 3 more

Open access · goldAbstract read
In one paragraph

Article in Frontiers in genetics, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
3.5field-weighted citation impact, top 7% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 28 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Article
  5. Review
  6. Article
  7. Article
  8. Article
  9. Maternal caffeine intake and DNA methylation in newborn cord blood.The American journal of clinical nutrition · 2022
    Article
  10. Article
  11. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors at 2 institutions in 2 countries.

Jamal S M SabirCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
Abdelfatteh El OmriCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
Babajan BanaganapalliDepartment of Genetic Medicine, Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.
Majed A Al-ShaeriCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
Naser A AlkenaniBiology-Zoology Division, Department of Biological Sciences, Faculty of Science, King Abdulaziz University, Jeddah, Saudi Arabia.
Mumdooh J SabirDepartment of Computer Sciences, Faculty of Computers and Information Technology, King Abdulaziz University, Jeddah, Saudi Arabia.
Nahid H HajrahCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
Houda ZrelliCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
Lukasz CieslaDepartment of Biological Sciences, Science and Engineering Complex, The University of Alabama, Tuscaloosa, AL, United States.
Khalidah K NasserDepartment of Medical Laboratory Technology, Faculty of Applied Medical Sciences, King Abdulaziz University, Jeddah, Saudi Arabia.
Ramu ElangoDepartment of Genetic Medicine, Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.
Noor Ahmad ShaikDepartment of Genetic Medicine, Faculty of Medicine, King Abdulaziz University, Jeddah, Saudi Arabia.
Muhummadh KhanCenter of Excellence in Bionanoscience Research, King Abdulaziz University, Jeddah, Saudi Arabia.
King Abdulaziz University · SAUniversity of Alabama · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Rheumatoid arthritis (RA) is a chronic synovial autoinflammatory disease that destructs the cartilage and bone, leading to disability. The functional regulation of major immunity-related pathways like nuclear factor kappa B (NF-κB), which is involved in the chronic inflammatory reactions underlying the development of RA, remains to be explored. Therefore, this study has adopted statistical and knowledge-based systemic investigations (like gene correlation, semantic similarity, and topological parameters based on graph theory) to study the gene expression status of NF-κB protein family (NK

Indexed as

auto-inflammatory diseasegene expressionGEONF-κBrheumatoid arthritis

Identifiers

PMID31824568
PMCPMC6879671
OpenAlexW2991480431

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.