Evidence map›Paper›PMID 31822999›Full record

ArticlePflugers Archiv : European journal of physiology2020

Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Ca

Janine Ebner, Michal Cagalinec, Helmut Kubista, Hannes Todt, Petra L Szabo, Attila Kiss, Bruno K Podesser, Henrietta Cserne Szappanos, Livia C Hool, Karlheinz Hilber and 1 more

Open access · hybridAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Pflugers Archiv : European journal of physiology, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
0.6field-weighted citation impact, top 26% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 7 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 3 countries.

Janine EbnerDepartment of Neurophysiology and-Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstraße 17, 1090, Vienna, Austria.
Michal CagalinecDepartment of Cellular Cardiology, Institute of Experimental Endocrinology, Biomedical Research Center, University Science Park for Biomedicine, Slovak Academy of Sciences, Bratislava, Slovakia.
Helmut KubistaDepartment of Neurophysiology and-Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstraße 17, 1090, Vienna, Austria.
Hannes TodtDepartment of Neurophysiology and-Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstraße 17, 1090, Vienna, Austria.
Petra L SzaboLudwig Boltzmann Cluster for Cardiovascular Research at the Center for Biomedical Research, Medical University of Vienna, Vienna, Austria.
Attila KissLudwig Boltzmann Cluster for Cardiovascular Research at the Center for Biomedical Research, Medical University of Vienna, Vienna, Austria.
Bruno K PodesserLudwig Boltzmann Cluster for Cardiovascular Research at the Center for Biomedical Research, Medical University of Vienna, Vienna, Austria.
Henrietta Cserne SzappanosSchool of Human Sciences, The University of Western Australia, Crawley, WA, 6009, Australia.
Livia C HoolSchool of Human Sciences, The University of Western Australia, Crawley, WA, 6009, Australia.
Karlheinz HilberDepartment of Neurophysiology and-Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstraße 17, 1090, Vienna, Austria. karlheinz.hilber@meduniwien.ac.at.ORCID 0000-0002-3033-0874
Xaver KoenigDepartment of Neurophysiology and-Pharmacology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstraße 17, 1090, Vienna, Austria.
Medical University of Vienna · ATLudwig Boltzmann Institute for Cardiovascular Research · ATThe University of Western Australia · AUSlovak Academy of Sciences · SK

Funding

Austrian Science Fund FWF P 30234Austrian Science Fund FWF P30234-B27
6 · The paper itself

Abstract

Neuronal nitric oxide synthase (nNOS) is considered a regulator of Ca

Indexed as

Action PotentialsCalcium SignalingAnimalsCalcium Channels, L-TypeCells, CulturedEnzyme InhibitorsFemaleHeart VentriclesMaleMiceMice, Inbred C57BLMyocytes, CardiacNitric Oxide DonorsNitric Oxide Synthase Type IIIOrnithineRatsCalcium Channels, L-TypeEnzyme InhibitorsL-type calcium channel alpha(1C)N(5)-(1-imino-3-butenyl)ornithineNitric Oxide DonorsNitric Oxide Synthase Type IIIOrnithineCalcium cyclingCav1.2 channel regulationNeuronal nitric oxide synthaseSingle-channel recordingsVentricular cardiomyocytesWhole cell patch clamp

Identifiers

PMID31822999
PMCPMC6960210
OpenAlexW2994694903

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.