ArticlePLoS genetics2019

A mutant form of Dmc1 that bypasses the requirement for accessory protein Mei5-Sae3 reveals independent activities of Mei5-Sae3 and Rad51 in Dmc1 filament stability.

Diedre Reitz et al.PubMed ↗Full text ↗Publisher ↗

No numbers read from the abstract.