ArticleWorld journal of clinical cases2019
Hypoparathyroidism with Fahr's syndrome: A case report and review of the literature.
Article in World journal of clinical cases, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
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Who cites it
9 citing papers in PubMed, 19 citations in OpenAlex.
- Hypoparathyroidism-related Fahr syndrome presenting with new-onset seizures: a case report.Journal of medical case reports · 2026Article
- Generalized Dystonia in Fahr's Syndrome Secondary to Idiopathic Hypoparathyroidism: Improvement After Metabolic Correction.Journal of clinical neurology (Seoul, Korea) · 2026Article
- Fahr's Syndrome Secondary to Hypoparathyroidism Presenting With Neurological and Cardiac Manifestations.Cureus · 2025Article
- Fahr's Syndrome: A Rare Case Presentation.Cureus · 2023Article
- Fahr's syndrome as a manifestation of autoimmune polyendocrine syndrome-1 and its unusual association with neuromyelitis optica spectrum disorder.Frontiers in neuroscience · 2023Review
- Article
- Rapidly Developing Cataract in Young Adult Patients: Always a Matter for Further Evaluation.Cureus · 2021Article
- Article
- From cranial nerve palsy to seizures-All the signs that lead to secondary Fahr's syndrome.Clinical case reports · 2021Article
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Authors and funding
3 authors at 1 institution in 1 country.
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No grant is acknowledged in the PubMed record.
Abstract
backgroundHypoparathyroidism with basal ganglia calcification is clinically rare. Here, we report a case of Fahr's syndrome due to hypoparathyroidism and review the literature in terms of etiology, clinical manifestation, diagnosis, and treatment. CASE SUMMARY: A 62-year-old man experienced repeated twitching of both hands in recent 10 years. On July 28, 2017, the patient was admitted to our hospital due to slow response and speech difficulties. On medical examinations, he had a positive Chvostek sign, while no Albright's hereditary osteodystrophy signs or history of neck surgery or radiation, and his family members had no similar medical history. Laboratory examinations revealed hypocalcemia, hyperphosphatemia, and low parathyroid hormone (PTH) levels. Computed tomography revealed basal ganglia calcification. Based on these investigations, a diagnosis of Fahr's syndrome due to hypoparathyroidism was suggested. After receiving intravenous calcium gluconate to relieve symptoms, the patient continued to take oral calcium carbonate and calcitriol for treatment.
conclusionThe possibility of hypoparathyroidism should be considered in patients with chronic hypocalcemia, recurrent tetany, and even neuropsychiatric symptoms. Hypoparathyroidism is a common cause of basal ganglia calcification. Therefore, it is recommended that blood calcium, phosphorus, and PTH levels should be measured in all individuals with basal ganglia calcification to exclude hypoparathyroidism.
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