Evidence map›Paper›PMID 31722977›Full record

ArticleJournal of cell science2019

Loss of Ca

Viviane Nascimento Da Conceicao, Yuyang Sun, Emily K Zboril, Jorge J De la Chapa, Brij B Singh

Open access · hybridAbstract read
In one paragraph

Article in Journal of cell science, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
2.6field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 30 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Article
  5. Article
  6. The p53 target DRAM1 modulates calcium homeostasis and ER stress by promoting contact between lysosomes and the ER through STIM1.Proceedings of the National Academy of Sciences of the United States of America · 2024
    Article
  7. Article
  8. Article
  9. The Molecular Heterogeneity of Store-Operated CaInternational journal of molecular sciences · 2023
    Review
  10. New Frontiers on ER Stress Modulation: Are TRP Channels the Leading Actors?International journal of molecular sciences · 2022
    Review
  11. Review
  12. Article
  13. Review
  14. Article
  15. Inhibition of CaFrontiers in physiology · 2022
    Article
  16. Article
  17. Article
  18. Review
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Viviane Nascimento Da ConceicaoDepartment of Periodontics, School of Dentistry, University of Texas Health San Antonio, San Antonio, TX 78229, USA.
Yuyang SunDepartment of Periodontics, School of Dentistry, University of Texas Health San Antonio, San Antonio, TX 78229, USA.
Emily K ZborilDepartment of Periodontics, School of Dentistry, University of Texas Health San Antonio, San Antonio, TX 78229, USA.
Jorge J De la ChapaDepartment of Comprehensive Dentistry, School of Dentistry, University of Texas Health San Antonio, San Antonio, TX 78229, USA.
Brij B SinghDepartment of Periodontics, School of Dentistry, University of Texas Health San Antonio, San Antonio, TX 78229, USA singhbb@uthscsa.edu.ORCID 0000-0003-0535-5997
The University of Texas Health Science Center at San Antonio · US

Funding

TISSUE CULTURE---COREP30CA054174 · NCI · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI Lei Zheng · 1991 to 2026
$59.1M
Institute for Integration of Medicine & Science: A Partnership to Improve HealthUL1TR001120 · NCATS · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI CLARK, ROBERT A, HARGREAVES, KENNETH M · 2013 to 2017
$16.5M
ROLE OF CASPASE-2 IN OSTEOCOLAST APOPTOSIS AGE-EePENDENT OSTEOPOROSISP01AG019316 · NIA · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI HERMAN, BRIAN A. · 2001 to 2011
$11.3M
TRPC1, Calcium, and Saliva SecretionR01DE017102 · NIDCR · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI Brij B Singh · 2006 to 2026
$7.5M
Epigenetic regulations in Sjogern's syndromeR01DE022765 · NIDCR · UNIVERSITY OF TEXAS HLTH SCIENCE CENTER · PI SINGH, BRIJ B · 2017 to 2021
$1.8M
NCATS NIH HHS UL1 TR001120NCI NIH HHS P30 CA054174NIA NIH HHS P01 AG019316NIDCR NIH HHS R01 DE017102NIDCR NIH HHS R01 DE022765
6 · The paper itself

Abstract

Activation of cellular stresses is associated with inflammation; however, the mechanisms are not well identified. Here, we provide evidence that loss of Ca

Indexed as

Endoplasmic Reticulum StressAnimalsCalciumCalcium ChannelsCell MembraneMacrophagesMaleMiceMice, Inbred C57BLORAI1 ProteinRAW 264.7 CellsStromal Interaction Molecule 1TRPC Cation ChannelsCalciumCalcium ChannelsORAI1 ProteinOrai1 protein, mouseStim1 protein, mouseStromal Interaction Molecule 1transient receptor potential cation channel, subfamily C, member 1TRPC Cation ChannelsCa2+ modulationER stressImmune activationSOCE channelsTRPC1

Identifiers

PMID31722977
PMCPMC10682644
OpenAlexW2989132394

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.