Evidence map›Paper›PMID 31681592›Full record

ArticleFrontiers in oncology2019

The Association of

Szymon Zmorzyński, Sylwia Popek-Marciniec, Aneta Szudy-Szczyrek, Magdalena Wojcierowska-Litwin, Iwona Korszeń-Pilecka, Sylwia Chocholska, Wojciech Styk, Marek Hus, Agata A Filip

Open access · goldAbstract read
In one paragraph

Article in Frontiers in oncology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 1 pooled it
8.2field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 1 synthesis or guideline pooled it, 38 citations in OpenAlex.

  1. Pooled it
  2. Review
  3. Article
  4. Article
  5. Review
  6. Article
  7. Article
  8. Analysis of Mutational Status ofInternational journal of molecular sciences · 2024
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  11. The Relationship ofJournal of clinical medicine · 2023
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  15. Article
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  17. Review
  18. A Network Pharmacology Approach to Explore the Potential Mechanisms of Huangqin-Baishao Herb Pair in Treatment of Cancer.Medical science monitor : international medical journal of experimental and clinical research · 2020
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 1 institution in 1 country.

Szymon ZmorzyńskiDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Sylwia Popek-MarciniecDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Aneta Szudy-SzczyrekChair and Department of Hematooncology and Bone Marrow Transplantation, Medical University of Lublin, Lublin, Poland.
Magdalena Wojcierowska-LitwinDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Iwona Korszeń-PileckaDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Sylwia ChocholskaChair and Department of Hematooncology and Bone Marrow Transplantation, Medical University of Lublin, Lublin, Poland.
Wojciech StykDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Marek HusChair and Department of Hematooncology and Bone Marrow Transplantation, Medical University of Lublin, Lublin, Poland.
Agata A FilipDepartment of Cancer Genetics With Cytogenetic Laboratory, Medical University of Lublin, Lublin, Poland.
Medical University of Lublin · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Oxidative stress and systemic inflammation are closely linked with increased risk of cancer development. Tumor necrosis factor alpha (TNF-α) is one of the pro-inflammatory cytokines. Glutathione S-transferases (GSTs) are enzymes involved in oxidative stress handling. Polymorphisms of genes encoding mentioned molecules may potentially influence the risk and the outcome in neoplastic diseases. Multiple myeloma (MM) is a hematological malignancy characterized by clonal, atypical plasma cell proliferation. In the present study we investigated the association of deletion polymorphisms in

Indexed as

apoptosisbortezomibGSTM1GSTT1polymorphismrs1800629rs361525TNF-alpha

Identifiers

PMID31681592
PMCPMC6798955
OpenAlexW2979723652

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.