Evidence map›Paper›PMID 31671764›Full record

ArticleInternational journal of molecular sciences2019

Anti-Inflammatory and Anti-Apoptotic Effects of Stybenpropol A on Human Umbilical Vein Endothelial Cells.

Li Zhang, Feifei Wang, Qing Zhang, Qiuming Liang, Shumei Wang, Minghua Xian, Feng Wang

Open access · goldAbstract read
In one paragraph

Article in International journal of molecular sciences, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.6field-weighted citation impact, top 32% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 18 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Anti-inflammatory therapy of atherosclerosis: focusing on IKKβ.Journal of inflammation (London, England) · 2023
    Review
  5. Review
  6. Experimental and therapeutic medicine · 2022
    Article
  7. Article
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Li ZhangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. riguangbaihe1990@126.com.
Feifei WangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. wangfeifei351@163.com.
Qing ZhangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. zqing0904@163.com.
Qiuming LiangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. QiumingLiang1021@163.com.
Shumei WangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. smwang1966@163.com.
Minghua XianKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. xmh360@163.com.
Feng WangKey Laboratory of Digital Quality Evaluation of Chinese Materia Medica of State Administration of TCM, School of Traditional Chinese Medicine, Guangdong Pharmaceutical University, Guangzhou 510006, China. wfeng1230@163.com.
Guangdong Pharmaceutical University · CN

Funding

the National Natural Science Foundation of China 81773884the Provincial Key Platforms and Major Scientific Research Projects of Guangdong Universities 2017KTSCX101
6 · The paper itself

Abstract

Inflammation is a key mediator in the progression of atherosclerosis (AS). Benzoinum, a resin secreted from the bark of

Indexed as

Anti-Inflammatory AgentsAtherosclerosisBasidiomycotabcl-2-Associated X ProteinBenzoinCaspase 9Cell Adhesion Molecule-1HumansHuman Umbilical Vein Endothelial CellsI-kappa B KinaseIntercellular Adhesion Molecule-1Interleukin-1betaInterleukin-8NF-KappaB Inhibitor alphaProto-Oncogene Proteins c-bcl-2Signal TransductionAnti-Inflammatory AgentsBAX protein, humanbcl-2-Associated X ProteinBCL2 protein, humanBenzoinCaspase 9Cell Adhesion Molecule-1ICAM1 protein, humanI-kappa B KinaseIntercellular Adhesion Molecule-1Interleukin-1betaInterleukin-8NF-KappaB Inhibitor alphaNFKBIA protein, humanProto-Oncogene Proteins c-bcl-2RELA protein, humanTranscription Factor RelATumor Necrosis Factor-alphaVascular Cell Adhesion Molecule-1atherosclerosisbenzoinumHUVECsNF-κB and caspase-9 signaling pathwaysstybenpropol A

Identifiers

PMID31671764
PMCPMC6862503
OpenAlexW2982500559

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.