Evidence map›Paper›PMID 31633153›Full record

ReviewEndocrine reviews2020

The New Biology of Diabetic Kidney Disease-Mechanisms and Therapeutic Implications.

Yuliya Lytvyn, Petter Bjornstad, Daniel H van Raalte, Hiddo L Heerspink, David Z I Cherney

Open access · bronzeAbstract readReview
In one paragraph

Review in Endocrine reviews, 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 87 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
87citing papers in PubMed, 3 pooled it
9.2field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

87 citing papers in PubMed, 3 syntheses or guidelines pooled it, 159 citations in OpenAlex.

  1. Pooled it
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  3. Pooled it
  4. Trial
  5. Ertugliflozin and Slope of Chronic eGFR: Prespecified Analyses from the Randomized VERTIS CV Trial.Clinical journal of the American Society of Nephrology : CJASN · 2021
    Trial
  6. Early Change in Albuminuria with Canagliflozin Predicts Kidney and Cardiovascular Outcomes: AJournal of the American Society of Nephrology : JASN · 2020 · on this map
    Trial
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  11. Review
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27 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 4 countries.

Yuliya LytvynDepartment of Medicine, Division of Nephrology, Toronto General Hospital, University of Toronto, Toronto, Ontario, Canada.
Petter BjornstadDepartment of Medicine, Division of Nephrology, Department of Pediatrics, Section of Endocrinology, University of Colorado School of Medicine, Aurora, Colorado.
Daniel H van RaalteDiabetes Center, Department of Internal Medicine, VU University Medical Center, Netherlands.
Hiddo L HeerspinkThe George Institute for Global Health, Sydney, Australia.
David Z I CherneyDepartment of Medicine, Division of Nephrology, Toronto General Hospital, University of Toronto, Toronto, Ontario, Canada.
University of Toronto · CAAmsterdam UMC Location VUmc · NLUniversity of Colorado Denver · USUniversity of Groningen · NL

Funding

Renal HEIR Study: Renal Hemodynamics, Energetics and Insulin Resistance in Youth Onset Type 2 Diabetes StudyK23DK116720 · NIDDK · UNIVERSITY OF COLORADO DENVER · PI BJORNSTAD, PETTER · 2018 to 2022
$963k
NIDDK NIH HHS K23 DK116720
6 · The paper itself

Abstract

Diabetic kidney disease remains the most common cause of end-stage kidney disease in the world. Despite reductions in incidence rates of myocardial infarction and stroke in people with diabetes over the past 3 decades, the risk of diabetic kidney disease has remained unchanged, and may even be increasing in younger individuals afflicted with this disease. Accordingly, changes in public health policy have to be implemented to address the root causes of diabetic kidney disease, including the rise of obesity and diabetes, in addition to the use of safe and effective pharmacological agents to prevent cardiorenal complications in people with diabetes. The aim of this article is to review the mechanisms of pathogenesis and therapies that are either in clinical practice or that are emerging in clinical development programs for potential use to treat diabetic kidney disease.

Indexed as

Glucagon-Like Peptide-1 Receptor AgonistsAnti-Inflammatory AgentsDiabetic NephropathiesDipeptidyl-Peptidase IV InhibitorsEndothelin Receptor AntagonistsHumansHypoglycemic AgentsMineralocorticoid Receptor AntagonistsSodium-Glucose Transporter 2 InhibitorsAnti-Inflammatory AgentsDipeptidyl-Peptidase IV InhibitorsEndothelin Receptor AntagonistsGlucagon-Like Peptide-1 Receptor AgonistsHypoglycemic AgentsMineralocorticoid Receptor AntagonistsSodium-Glucose Transporter 2 Inhibitors

Identifiers

PMID31633153
PMCPMC7156849
OpenAlexW2981496710

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.