Evidence map›Paper›PMID 31605649›Full record

ArticleMicrocirculation (New York, N.Y. : 1994)2020

Hyperinsulinemia does not cause de novo capillary recruitment in rat skeletal muscle.

Thorbjorn Akerstrom, Daniel Goldman, Franciska Nilsson, Stephanie L Milkovich, Graham M Fraser, Christian Lehn Brand, Ylva Hellsten, Christopher G Ellis

Open access · hybridAbstract read
In one paragraph

Article in Microcirculation (New York, N.Y. : 1994), 2020. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.3field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 20 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 2 countries.

Thorbjorn AkerstromDepartment of Nutrition, Exercise and Sports, Section of Integrative Physiology, University of Copenhagen, Copenhagen, Denmark.
Daniel GoldmanDepartment of Medical Biophysics, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.ORCID 0000-0002-8707-5536
Franciska NilssonDepartment of Nutrition, Exercise and Sports, Section of Integrative Physiology, University of Copenhagen, Copenhagen, Denmark.
Stephanie L MilkovichDepartment of Medical Biophysics, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.
Graham M FraserDivision of BioMedical Sciences, Faculty of Medicine, Memorial University of Newfoundland, St. John's, Canada.ORCID 0000-0002-5733-201X
Christian Lehn BrandClamp Competency Centre, Novo Nordisk A/S, Maaloev, Denmark.
Ylva HellstenDepartment of Nutrition, Exercise and Sports, Section of Integrative Physiology, University of Copenhagen, Copenhagen, Denmark.ORCID 0000-0002-2435-9558
Christopher G EllisDepartment of Medical Biophysics, Schulich School of Medicine & Dentistry, University of Western Ontario, London, Canada.ORCID 0000-0002-7760-1846
University of Copenhagen · DKWestern University · CAMemorial University of Newfoundland · CANovo Nordisk (Denmark) · DK

Funding

CIHR PJT-162119
6 · The paper itself

Abstract

objectiveThe effect of insulin on blood flow distribution within muscle microvasculature has been suggested to be important for glucose metabolism. However, the "capillary recruitment" hypothesis is still controversial and relies on studies using indirect contrast-enhanced ultrasound (CEU) methods.

methodsWe studied how hyperinsulinemia effects capillary blood flow in rat extensor digitorum longus (EDL) muscle during euglycemic hyperinsulinemic clamp using intravital video microscopy (IVVM). Additionally, we modeled blood flow and microbubble distribution within the vascular tree under conditions observed during euglycemic hyperinsulinemic clamp experiments.

resultsEuglycemic hyperinsulinemia caused an increase in erythrocyte (80 ± 25%, P < .01) and plasma (53 ± 12%, P < .01) flow in rat EDL microvasculature. We found no evidence of de novo capillary recruitment within, or among, capillary networks supplied by different terminal arterioles; however, erythrocyte flow became slightly more homogenous. Our computational model predicts that a decrease in asymmetry at arteriolar bifurcations causes redistribution of microbubble flow among capillaries already perfused with erythrocytes and plasma, resulting in 25% more microbubbles flowing through capillaries.

conclusionsOur model suggests increase in CEU signal during hyperinsulinemia reflects a redistribution of arteriolar flow and not de novo capillary recruitment. IVVM experiments support this prediction showing increases in erythrocyte and plasma flow and not capillary recruitment.

Indexed as

CapillariesHyperinsulinismMicrocirculationMuscle, SkeletalAnimalsMaleRatsRats, Sprague-Dawleyinsulinmicrocirculationskeletal muscle

Identifiers

PMID31605649
PMCPMC7064932
OpenAlexW2979386122

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.