Evidence map›Paper›PMID 31601780›Full record

ArticleCell death & disease2019

Ca

Hao-Song Shi, Ke Lai, Xin-Lu Yin, Min Liang, Hai-Bo Ye, Hai-Bo Shi, Lu-Yang Wang, Shan-Kai Yin

Open access · goldAbstract read
In one paragraph

Article in Cell death & disease, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
1.8field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Article
  3. Selective Vulnerability of GABAergic Inhibitory Interneurons to Bilirubin Neurotoxicity in the Neonatal Brain.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2024
    Article
  4. Article
  5. Article
  6. Review
  7. Article
  8. Downregulation of CaFrontiers in pharmacology · 2022
    Article
  9. Article
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 2 countries.

Hao-Song ShiDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Ke LaiDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Xin-Lu YinDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Min LiangDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Hai-Bo YeDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Hai-Bo ShiDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China. hbshi@sjtu.edu.cn.
Lu-Yang WangPrograms in Neurosciences & Mental Health, SickKids Research Institute and Department of Physiology, University of Toronto, Toronto, ON, M5G 1X8, Canada. luyang.wang@utoronto.ca.
Shan-Kai YinDepartment of Otorhinolaryngology, The Sixth People's Hospital of Shanghai and Shanghai Jiao Tong University, 600 Yishan Road, 200233, Shanghai, P. R. China.
Shanghai Jiao Tong University · CNShanghai Sixth People's Hospital · CNUniversity of Toronto · CA

Funding

CIHR MOP-77610CIHR MOP-81159CIHR PJT-156034CIHR PJT-156439
6 · The paper itself

Abstract

Neonatal jaundice is prevalent among newborns and can lead to severe neurological deficits, particularly sensorimotor dysfunction. Previous studies have shown that bilirubin (BIL) enhances the intrinsic excitability of central neurons and this can potentially contribute to their overexcitation, Ca

Indexed as

Action PotentialsAnimalsBilirubinCalciumCell DeathExocytosisNAV1.1 Voltage-Gated Sodium ChannelNeuronsRatsRats, Sprague-DawleyUp-RegulationVestibular NucleiVoltage-Gated Sodium ChannelsBilirubinCalciumNAV1.1 Voltage-Gated Sodium ChannelScn1a protein, ratVoltage-Gated Sodium Channels

Identifiers

PMID31601780
PMCPMC6787254
OpenAlexW2980241471

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.