Evidence map›Paper›PMID 31577254›Full record

ReviewActa bio-medica : Atenei Parmensis2019

Monogenic hypertension.

Vincenza Precone, Geraldo Krasi, Giulia Guerri, Liborio Stuppia, Francesco Romeo, Marco Perrone, Carla Marinelli, Alessandra Zulian, Tiziano Dallavilla, Matteo Bertelli

Abstract readReview
In one paragraph

Review in Acta bio-medica : Atenei Parmensis, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Vincenza PreconeMAGI Euregio, Bolzano, Italy. vincenza_precone@yahoo.it.
Geraldo Krasi
Giulia Guerri
Liborio Stuppia
Francesco Romeo
Marco Perrone
Carla Marinelli
Alessandra Zulian
Tiziano Dallavilla
Matteo Bertelli

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hypertension is a significant public health problem. Thirty percent of cases are caused by a single genetic mutation. Hypertension is the predominant and usually the only manifestation in monogenic hypertension Monogenic hypertension may involve mineralcorticoid-dependent or -independent increase in Na+ transport. Diagnosis is based on routine physical examination, blood pressure measurement and laboratory analysis of renin, aldosterone, cortisol and potassium. Genetic testing is useful for confirming diagnosis and for differential diagnosis. Monogenic hypertension has autosomal dominant or autosomal recessive inheritance.

Indexed as

AldosteroneGenetic TestingHigh-Throughput Nucleotide SequencingHumansHydrocortisoneHypertensionMutationPotassiumReninAldosteroneHydrocortisonePotassiumRenin

Identifiers

PMID31577254
PMCPMC7233634

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.