Evidence map›Paper›PMID 31576093›Full record

ArticleWorld journal of gastroenterology2019

Qin Zhao, Wen-Rong Yang, Xiao-Hong Wang, Gai-Qin Li, Lei-Qi Xu, Xiao Cui, Yang Liu, Xiu-Li Zuo

Open access · hybridAbstract read
In one paragraph

Article in World journal of gastroenterology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 39 papers.

0numbers the graph read from it
0cells of the map it votes in
39citing papers in PubMed
3.8field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

39 citing papers in PubMed, 73 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
  5. Article
  6. Current research in microbial sciences · 2026
    Article
  7. Article
  8. Article
  9. Frontiers in microbiology · 2025
    Article
  10. Article
  11. Article
  12. Article
  13. Review
  14. Article
  15. Review
  16. Article
  17. Article
  18. Frontiers in microbiology · 2024
    Article
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 2 institutions in 1 country.

Qin ZhaoDepartment of Gastroenterology, Qilu Hospital, Shandong University, Jinan 250012, Shandong Province, China.
Wen-Rong YangDepartment of Clinical Nutrition, Taian City Central Hospital, Taian 271000, Shandong Province, China.
Xiao-Hong WangDepartment of Gastroenterology, Taian City Central Hospital, Taian 271000, Shandong Province, China.
Gai-Qin LiDepartment of Gastroenterology, Taian City Central Hospital, Taian 271000, Shandong Province, China.
Lei-Qi XuDepartment of Gastroenterology, Qilu Hospital, Shandong University, Jinan 250012, Shandong Province, China.
Xiao CuiDepartment of Gastroenterology, Qilu Hospital, Shandong University, Jinan 250012, Shandong Province, China.
Yang LiuDepartment of Medicine, Beijing 316 Hospital, Beijing 100093, China.
Xiu-Li ZuoDepartment of Gastroenterology, Qilu Hospital, Shandong University, Jinan 250012, Shandong Province, China. zuoxiuli_s@163.com.
Qilu Hospital of Shandong University · CNTaian City Central Hospital · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundIrritable bowel syndrome (IBS) is one of the most common functional gas-troenterological diseases characterized by abnormal visceral sensitivity and low-grade inflammation. The role of

aimTo investigate the role of DCs in the development of IBS in a rat model and to understand the regulation of DCs after

methodsAn IBS animal model was established using C57BL/6 mice, and

resultsThe secretion of proinflammatory cytokines (IL-1β and IL-6) in mice with IBS was significantly increased compared with that of the control group, which suggested that the intestinal mucosa in mice with IBS was in a low-grade inflammatory state. The expression of CD11C+CD80+ and CD11c+TIM3+ in intestinal LPDCs in mice with IBS increased significantly. Meanwhile, the cytokines (IL-1β and IL-6) were significantly reduced after the intervention with probiotic

conclusionThe results suggest that

Indexed as

AnimalsClostridium butyricumDendritic CellsDisease Models, AnimalHumansImmunity, MucosalIntestinal MucosaIrritable Bowel SyndromeMaleMiceMice, Inbred C57BLProbioticsTreatment OutcomeTrinitrobenzenesulfonic AcidTrinitrobenzenesulfonic AcidClostridium butyricumIrritable bowel syndromeLamina propria dendritic cellsProinflammatory cytokinesT cell immunoglobulin and mucin domain-3

Identifiers

PMID31576093
PMCPMC6767978
OpenAlexW2976418724

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.