Evidence map›Paper›PMID 31564230›Full record

ArticleCell cycle (Georgetown, Tex.)2019

Dysfunctional CAF-I reveals its role in cell cycle progression and differential regulation of gene silencing.

Hollie Rowlands, Kholoud Shaban, Ashley Cheng, Barret Foster, Krassimir Yankulov

Open access · bronzeAbstract read
In one paragraph

Article in Cell cycle (Georgetown, Tex.), 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.2field-weighted citation impact, top 47% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Hollie RowlandsDepartment of Molecular and Cellular Biology, University of Guelph , Guelph , Canada.ORCID https://orcid.org/0000-0003-2650-0962
Kholoud ShabanDepartment of Molecular and Cellular Biology, University of Guelph , Guelph , Canada.
Ashley ChengDepartment of Molecular and Cellular Biology, University of Guelph , Guelph , Canada.
Barret FosterDepartment of Molecular and Cellular Biology, University of Guelph , Guelph , Canada.
Krassimir YankulovDepartment of Molecular and Cellular Biology, University of Guelph , Guelph , Canada.ORCID https://orcid.org/0000-0001-9440-9234
University of Guelph · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chromatin Assembly Factor I (CAF-I) plays a central role in the reassembly of H3/H4 histones during DNA replication. In

Indexed as

Gene SilencingCDC2 Protein KinaseCell CycleCell Cycle ProteinsCell DivisionChromatinChromatin Assembly Factor-1DNA DamageDNA ReplicationGenes, Mating Type, FungalMannose-Binding LectinsMutationPhenotypePhosphorylationProliferating Cell Nuclear AntigenProtein Serine-Threonine KinasesCDC2 Protein KinaseCDC7 protein, S cerevisiaeCell Cycle ProteinsChromatinChromatin Assembly Factor-1FLO1 protein, S cerevisiaeMannose-Binding LectinsPOL30 protein, S cerevisiaeProliferating Cell Nuclear AntigenProtein Serine-Threonine KinasesRLF2 protein, S cerevisiaeSaccharomyces cerevisiae ProteinsSilent Information Regulator Proteins, Saccharomyces cerevisiaeSIR2 protein, S cerevisiaeSirtuin 2cell cycleChromatin assembly factor Icyclin dependent kinase (CDK)Dbf4-dependent kinase (DDK)flocculationgenes

Identifiers

PMID31564230
PMCPMC6816422
OpenAlexW2977556245

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.