ArticleCaspian journal of internal medicine2019
Libman-Sacks endocarditis in patients with systemic lupus erythematosus with secondary antiphospholipid syndrome.
Article in Caspian journal of internal medicine, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.
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Who cites it
9 citing papers in PubMed, 12 citations in OpenAlex.
- Systemic Lupus Erythematosus With Catastrophic APS and Libman-Sacks Endocarditis in a Patient With Autoimmune Hemolytic Anemia: A Case Report.Clinical case reports · 2025Article
- Left Ventricle Libman-Sacks Endocarditis Secondary to Systemic Lupus Erythematosus and Antiphospholipid Syndrome: A Case Report.The American journal of case reports · 2025Article
- A rare case report of coexisting rheumatic heart disease and systemic lupus erythematous.Clinical case reports · 2024Article
- Exploring cardiovascular implications in systemic lupus erythematosus: A holistic analysis of complications, diagnostic criteria, and therapeutic modalities, encompassing pharmacological and adjuvant approaches.Biomolecular concepts · 2024Review
- Article
- Ischemic Stroke Revealing Libman-Sacks Endocarditis: A Case Report.Journal of the Saudi Heart Association · 2023Article
- Article
- Review
- Multiple embolic strokes in primary antiphospholipid syndrome.European journal of rheumatology · 2020Article
Corrections and comments
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundLibman-Sacks endocarditis (LSE) is characterized by sterile lesions that commonly affect the aortic and mitral heart valves. Antiphospholipid syndrome (APS) and systemic lupus erythematosus (SLE) have been associated with LSE. Cardiac manifestations including LSE could be interrelated with other manifestations and early diagnosis could help in preventing further complications. CASE PRESENTATION: Here, we report three cases of LSE in SLE patients with secondary APS. All patients presented with neurological manifestations and LSE was diagnosed by Transesophageal echocardiography (TEE). All three patients were treated for the underlying disease and also received anticoagulant therapy.
conclusionIn all patients with SLE and secondary APS, LSE should be considered if a patient manifests any evidence of neurologic involvement.
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Registered trials
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