Evidence map›Paper›PMID 31554702›Full record

ReviewEuropean respiratory review : an official journal of the European Respiratory Society2019

The genetics of interstitial lung diseases.

Raphael Borie, Pierre Le Guen, Mada Ghanem, Camille Taillé, Clairelyne Dupin, Philippe Dieudé, Caroline Kannengiesser, Bruno Crestani

Open access · diamondAbstract readReview
In one paragraph

Review in European respiratory review : an official journal of the European Respiratory Society, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 37 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
37citing papers in PubMed, 2 pooled it
5.1field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

37 citing papers in PubMed, 2 syntheses or guidelines pooled it, 59 citations in OpenAlex.

  1. Pooled it
  2. Pooled it
  3. Article
  4. Review
  5. Interstitial lung disease and the STING pathway.The Journal of clinical investigation · 2026
    Review
  6. Article
  7. Article
  8. Article
  9. Article
  10. Review
  11. Article
  12. Review
  13. Article
  14. Review
  15. Article
  16. Article
  17. Proprotein Convertase Subtilisin/Kexin Type 6: A Risk Factor for Survival in Pulmonary Fibrosis?American journal of respiratory and critical care medicine · 2023
    Article
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Raphael BorieService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France.ORCID https://orcid.org/0000-0002-9906-0024
Pierre Le GuenService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France.
Mada GhanemService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France.
Camille TailléService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France.
Clairelyne DupinService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France.ORCID https://orcid.org/0000-0001-7957-8195
Philippe DieudéINSERM U1152, Paris, France.ORCID https://orcid.org/0000-0002-4814-0307
Caroline KannengiesserINSERM U1152, Paris, France.ORCID https://orcid.org/0000-0003-2155-9383
Bruno CrestaniService de Pneumologie A, Hôpital Bichat, AP-HP, Paris, France bruno.crestani@aphp.fr.
Inserm · FR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Interstitial lung diseases (ILDs) are a set of heterogeneous lung diseases characterised by inflammation and, in some cases, fibrosis. These lung conditions lead to dyspnoea, cough, abnormalities in gas exchange, restrictive physiology (characterised by decreased lung volumes), hypoxaemia and, if progressive, respiratory failure. In some cases, ILDs can be caused by systemic diseases or environmental exposures. The ability to treat or cure these ILDs varies based on the subtype and in many cases lung transplantation remains the only curative therapy. There is a growing body of evidence that both common and rare genetic variants contribute to the development and clinical manifestation of many of the ILDs. Here, we review the current understanding of genetic risk and ILD.

Indexed as

Genetic VariationAnimalsGenetic Predisposition to DiseaseHumansLungLung Diseases, InterstitialLung TransplantationPhenotypeRisk AssessmentRisk FactorsTreatment Outcome

Identifiers

PMID31554702
PMCPMC9488931
OpenAlexW2976980646

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.