Evidence map›Paper›PMID 31548372›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2019

Dronc-independent basal executioner caspase activity sustains

Natsuki Shinoda, Nozomi Hanawa, Takahiro Chihara, Akiko Koto, Masayuki Miura

Open access · bronzeAbstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed
2.6field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 68 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 3 institutions in 1 country.

Natsuki ShinodaDepartment of Genetics, Graduate School of Pharmaceutical Sciences, The University of Tokyo, Bunkyo-ku, 113-0033 Tokyo, Japan.
Nozomi HanawaDepartment of Genetics, Graduate School of Pharmaceutical Sciences, The University of Tokyo, Bunkyo-ku, 113-0033 Tokyo, Japan.
Takahiro ChiharaDepartment of Biological Science, Graduate School of Science, Hiroshima University, Higashi-Hiroshima, 739-8526 Hiroshima, Japan.ORCID 0000-0001-9989-3619
Akiko KotoBioproduction Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Tsukuba, 305-8566 Ibaraki, Japan.
Masayuki MiuraDepartment of Genetics, Graduate School of Pharmaceutical Sciences, The University of Tokyo, Bunkyo-ku, 113-0033 Tokyo, Japan; miura@mol.f.u-tokyo.ac.jp.
The University of Tokyo · JPHiroshima University · JPWaseda University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Caspase is best known as an enzyme involved in programmed cell death, which is conserved among multicellular organisms. In addition to its role in cell death, caspase is emerging as an indispensable enzyme in a wide range of cellular functions, which have recently been termed caspase-dependent nonlethal cellular processes (CDPs). In this study, we examined the involvement of cell death signaling in tissue-size determination using

Indexed as

AnimalsApoptosisCaspasesDrosophila melanogasterDrosophila ProteinsImaginal DiscsWings, AnimalCaspasesDcp-1 protein, Drosophiladecay protein, Drosophiladronc protein, DrosophilaDrosophila Proteinscaspase-dependent nonlethal cellular processesexecutioner caspasefluctuating asymmetrytissue-size regulationTurboID

Identifiers

PMID31548372
PMCPMC6789915
OpenAlexW2976301373

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.