Evidence map›Paper›PMID 31534958›Full record

ArticleBioMed research international2019

FcgRIII Deficiency and FcgRIIb Defeciency Promote Renal Injury in Diabetic Mice.

Rui Zhang, Tingli Wang, Qinhua Yin, Junlin Zhang, Li Li, Ruikun Guo, Qianqian Han, Hanyu Li, Yiting Wang, Jiali Wang and 6 more

Erratum issuedOpen access · hybridAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in BioMed research international, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.5field-weighted citation impact, top 31% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors at 1 institution in 1 country.

Rui ZhangDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Tingli WangDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Qinhua YinDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Junlin ZhangDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Li LiDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Ruikun GuoDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Qianqian HanDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Hanyu LiDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Yiting WangDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Jiali WangDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Pramesh GurungDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Yanrong LuKey Laboratory of Transplant Engineering and Immunology, Ministry of Health, Regenerative Medicine Research Center, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Jingqiu ChengKey Laboratory of Transplant Engineering and Immunology, Ministry of Health, Regenerative Medicine Research Center, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Lin BaiKey Laboratory of Transplant Engineering and Immunology, Ministry of Health, Regenerative Medicine Research Center, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Jie ZhangKey Laboratory of Transplant Engineering and Immunology, Ministry of Health, Regenerative Medicine Research Center, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.
Fang LiuDivision of Nephrology, West China Hospital of Sichuan University, Chengdu 610041, Sichuan, China.ORCID https://orcid.org/0000-0003-1121-3004
Sichuan University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The immune system is involved in the development of diabetes complications and IgG Fc gamma receptors (FcgRs) are key immune receptors responsible for the effective control of both humoral and innate immunity. We investigated the effects of members of the FcgR superfamily into both the streptozotocin plus high fat-induced type 2 diabetes and high fat diet (HFD) models. FcgRIII

Indexed as

AnimalsDiabetes Mellitus, Type 2Diabetic NephropathiesDietary FatsLipoproteins, LDLMiceMice, KnockoutReceptors, IgGTranscription Factor RelATransforming Growth Factor beta1Tumor Necrosis Factor-alphaDietary FatsFcgr2b protein, mouseFcgr3 protein, mouseLipoproteins, LDLoxidized low density lipoproteinReceptors, IgGRela protein, mouseTgfb1 protein, mouseTranscription Factor RelATransforming Growth Factor beta1Tumor Necrosis Factor-alpha

Identifiers

PMID31534958
PMCPMC6724446
OpenAlexW2969711717

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.