Evidence map›Paper›PMID 31463128›Full record

ArticleJournal of thoracic disease2019

Clinical importance of

Kazutoshi Isobe, Takuma Issiki, Susumu Sakamoto, Go Sano, Yujiro Takai, Naobumi Tochigi, Sakae Homma

Open access · diamondAbstract read
In one paragraph

Article in Journal of thoracic disease, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.5field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Kazutoshi IsobeDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Takuma IssikiDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Susumu SakamotoDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Go SanoDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Yujiro TakaiDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Naobumi TochigiDivision of Surgical Pathology, Toho University School of Medicine, Tokyo, Japan.
Sakae HommaDivision of Respiratory Medicine, Toho University School of Medicine, Tokyo, Japan.
Toho University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundReactive oxygen species (ROS) can play a role in the pathogenesis of Idiopathic Pulmonary Fibrosis (IPF) by contributing to epithelial damage.

methodsIn this single-center retrospective study, we reviewed the medical records of 63 patients with IPF who were treated at our hospital from January 2006 through April 2018. Patients with and without

resultsNo fatal AE-IPF occurred in patients with

conclusionsIPF patients with

Indexed as

acute exacerbation (AE)Bcl-2-like 11 (BIM) deletion polymorphismIdiopathic pulmonary fibrosis (IPF)interstitial pneumonia (IP)usual interstitial pneumonia (UIP)

Identifiers

PMID31463128
PMCPMC6688017
OpenAlexW2962980068

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.