Evidence map›Paper›PMID 31439126›Full record

ArticleeLife2019

Two-step regulation of

Takefumi Kondo, Shigeo Hayashi

Abstract read
In one paragraph

Article in eLife, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Pri micropeptide functions as a cell-intrinsic timer controlling the transient phase of cell fate induction.Proceedings of the National Academy of Sciences of the United States of America · 2026
    Article
  2. The dynamics of tubulogenesis in development and disease.Development (Cambridge, England) · 2025
    Review
  3. Article
  4. Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Takefumi KondoGraduate School of Biostudies, Kyoto University, Kyoto, Japan.ORCID 0000-0001-8127-1141
Shigeo HayashiLaboratory for Morphogenetic Signaling, RIKEN Center for Biosystems Dynamics Research, Kobe, Japan.ORCID 0000-0001-7785-1290

Funding

Japan Agency for Medical Research and Development JP18gm5810020Japan Society for the Promotion of Science 15H05597Ministry of Education, Culture, Sports, Science, and Technology Building of Consortia for the Development of Human Resources in Science and Technology
6 · The paper itself

Abstract

During organogenesis, inductive signals cause cell differentiation and morphogenesis. However, how these phenomena are coordinated to form functional organs is poorly understood. Here, we show that cell differentiation of the

Indexed as

Gene Expression Regulation, DevelopmentalMorphogenesisAnimalsCell DifferentiationDrosophilaDrosophila ProteinsEpithelial CellsTracheaTranscription FactorsDrosophila ProteinsTranscription Factorstrh protein, Drosophilacell differentiationdevelopmental biologyD. melanogasterDrosophilaepithelial morphogenesisgene expressioninvaginationtrachea

Identifiers

PMID31439126
PMCPMC6707767

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.