Evidence map›Paper›PMID 31375581›Full record

ArticleJournal of virology2019

Endosomal Toll-Like Receptors Mediate Enhancement of Interleukin-17A Production Triggered by Epstein-Barr Virus DNA in Mice.

Marwa Shehab, Nour Sherri, Hadi Hussein, Noor Salloum, Elias A Rahal

Open access · bronzeAbstract read
In one paragraph

Article in Journal of virology, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
1.3field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed, 28 citations in OpenAlex.

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  17. Observational
  18. Frontiers in immunology · 2021
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 1 institution in 1 country.

Marwa ShehabDepartment of Experimental Pathology, Immunology and Microbiology, American University of Beirut, Beirut, Lebanon.
Nour SherriDepartment of Experimental Pathology, Immunology and Microbiology, American University of Beirut, Beirut, Lebanon.
Hadi HusseinDepartment of Experimental Pathology, Immunology and Microbiology, American University of Beirut, Beirut, Lebanon.
Noor SalloumDepartment of Experimental Pathology, Immunology and Microbiology, American University of Beirut, Beirut, Lebanon.
Elias A RahalDepartment of Experimental Pathology, Immunology and Microbiology, American University of Beirut, Beirut, Lebanon er00@aub.edu.lb.ORCID 0000-0002-1096-7042
American University of Beirut · LB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

We previously demonstrated that Epstein-Barr virus (EBV) DNA increases the production of the proinflammatory cytokine interleukin-17A (IL-17A) in mice. This property may contribute to the established association between EBV and autoimmune diseases. The objective of the present study was to elucidate mechanisms through which EBV DNA modulates IL-17A levels in mice. To determine whether endosomal Toll-like receptors (TLRs) played a role in this pathway, the expression of TLR3, -7, or -9 was assessed by real-time reverse transcription-PCR in mouse spleens after injection of EBV DNA. Moreover, specific inhibitors were used for these TLRs in mouse peripheral blood mononuclear cells (PBMCs) cultured with EBV DNA and in mice injected with this viral DNA; IL-17A levels were then assessed using an enzyme-linked immunosorbent assay. The expression of the endosomal receptors TLR3, -7, and -9 was increased in mice injected with EBV DNA. When mouse immune cells were cultured with EBV DNA and a TLR3, -7, or -9 inhibitor or when mice were injected with the viral DNA along with either of these inhibitors, a significant decrease in IL-17A levels was detected. Therefore, endosomal TLRs are involved in the EBV DNA-mediated triggering of IL-17A production in mice. Targeting these receptors in EBV-positive subjects with autoimmunity may be useful pending investigations assessing whether they play a similar role in humans.

Indexed as

AnimalsDNA, ViralEndosomesEpstein-Barr Virus InfectionsHerpesvirus 4, HumanHumansInterleukin-17Leukocytes, MononuclearMiceSpleenToll-Like ReceptorsDNA, ViralIL17A protein, humanInterleukin-17Toll-Like ReceptorsEpstein-Barr virushuman herpesvirusesIL-17AinterleukinsTLR3TLR7TLR9Toll-like receptors

Identifiers

PMID31375581
PMCPMC6798095
OpenAlexW2964551630

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.