Evidence map›Paper›PMID 31354195›Full record

ArticleBioinformation2019

Impact of rotavirus and hepatitis A virus by worldwide climatic changes during the period between 2000 and 2013.

Fatima Tarek, Najwa Hassou, Mohammed Nabil Benchekroun, Said Boughribil, Jamal Hafid, My Mustapha Ennaji

Open access · hybridAbstract read
In one paragraph

Article in Bioinformation, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.6field-weighted citation impact, top 33% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 10 citations in OpenAlex.

  1. Climate change and liver disease: a mini review.Frontiers in gastroenterology (Lausanne, Switzerland) · 2024
    Review
  2. Article
  3. Article
  4. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Fatima TarekTeam of Virology and Oncology, Laboratory of Virology, Microbiology, Quality and Biotechnology/Ecotoxicology and Biodiversity, Faculty of Sciences and Techniques Mohammedia, University Hassan II of Casablanca.
Najwa HassouTeam of Virology and Oncology, Laboratory of Virology, Microbiology, Quality and Biotechnology/Ecotoxicology and Biodiversity, Faculty of Sciences and Techniques Mohammedia, University Hassan II of Casablanca.
Mohammed Nabil BenchekrounTeam of Biotechnology an Environment Laboratory of Virology, Microbiology, Quality and Biotechnology/ Eco toxicology and Biodiversity, Faculty of Sciences and techniques Mohammedia,University Hassan II of Casablanca.
Said BoughribilTeam of Eco toxicology and Biodiversity, Laboratory of Virology, Microbiology, Quality and Biotechnology/Ecotoxicology and Biodiversity, Faculty of Sciences and techniques Mohammedia, University Hassan II of Casablanca.
Jamal HafidTeam of Immuno parasitology, Laboratory food, Environment and Health FST Gueliz, University Cadi Ayyad Marrakech.
My Mustapha EnnajiTeam of Virology and Oncology, Laboratory of Virology, Microbiology, Quality and Biotechnology/Ecotoxicology and Biodiversity, Faculty of Sciences and Techniques Mohammedia, University Hassan II of Casablanca.
University of Hassan II Casablanca · MACadi Ayyad University · MAUniversité Hassan II Mohammedia · MA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Enteric viruses are present in the environment as a result of the discharge of poorly or untreated wastewater. The spread of enteric viruses in the environment depend to human activities like stools of infected individuals ejected in the external environment can be transmitted by water sources and back to susceptible individuals for other cycles of illness. Among the enteric viruses Rotaviruses (RV) and Hepatitis A viruses (HAV) is the most detected in wastewater causing gastroenteritis and acute hepatitis. Therefore, it is of interest to climate change, mainly temperature and carbon Dioxide (CO2) variations, on Rotavirus and Hepatitis A as a model of enteric viruses present in the aquatic environment using computational modelling tools. The results of genetic ratio showed a negative correlation between the epidemiological data and the mutation rate. However, the correlation was positive between the temperature, CO2 increase, and the rate of mutation. The positive correlation is explained by the adaptation of the viruses to the climatic changes, the RNA polymerase of the RV induces errors to adapt to the environmental conditions. The simultaneous increase in number of infections and temperature in 2010 has been demonstrated in previous studies deducing that viral pathogenicity increase with temperature increase.

Indexed as

Carbon dioxidehepatitis A virusmutation raterotavirustemperature variations

Identifiers

PMID31354195
PMCPMC6637397
OpenAlexW2955256573

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.