Evidence map›Paper›PMID 31308803›Full record

ReviewJournal of ginseng research2019

Ameliorative effects of ginseng and ginsenosides on rheumatic diseases.

Young-Su Yi

Open access · goldAbstract readReview
In one paragraph

Review in Journal of ginseng research, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
1.7field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 44 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
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  5. Review
  6. Article
  7. Article
  8. Review
  9. Article
  10. Review
  11. Article
  12. Anti-fatigue effect fromAcupuncture and herbal medicine · 2022
    Review
  13. Article
  14. Article
  15. Ginsenoside Ro, an oleanolic saponin ofJournal of ginseng research · 2022
    Article
  16. Review
  17. Review
  18. Review
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Young-Su YiDepartment of Pharmaceutical Engineering, Cheongju University, Cheongju, Republic of Korea.
Cheongju University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundInflammation is a host-defensive innate immune response to protect the body from pathogenic agents and danger signals induced by cellular changes. Although inflammation is a host-defense mechanism, chronic inflammation is considered a major risk factor for the development of a variety of inflammatory autoimmune diseases, such as rheumatic diseases. Rheumatic diseases are systemic inflammatory and degenerative diseases that primarily affect connective tissues and are characterized by severe chronic inflammation and degeneration of connective tissues. Ginseng and its bioactive ingredients, genocides, have been demonstrated to have antiinflammatory activity and pharmacological effects on various rheumatic diseases by inhibiting the expression and production of inflammatory mediators.

methodsLiterature in this review was searched in a PubMed site of National Center for Biotechnology Information.

resultsThe studies reporting the preventive and therapeutic effects of ginseng and ginsenosides on the pathogenesis of rheumatic diseases were discussed and summarized.

conclusionGinseng and ginsenosides play an ameliorative role on rheumatic diseases, and this review provides new insights into ginseng and ginsenosides as promising agents to prevent and treat rheumatic diseases.

Indexed as

ACAN, AggrecanACLT, Anterior cruciate ligament transectionBMP, Bone morphogenetic proteinCIA, Collagen-induced arthriticCK, Compound KCOL, CollagenDAMP, Danger-associated molecular patternGinsengGinsenosidesInflammationLTMMR, Ligament transection and medial meniscus resectionMacrophagesOA, OsteoarthritisPAMP, Pathogen-associated molecular patternPPD, ProtopanaxadiolPPT, ProtopanaxatriolPRR, Pattern-recognition receptorRA, Rheumatoid arthritisRheumatic diseasesRNAKL, Receptor activator of NF-κB ligandSLE, Systemic lupus erythematosus

Identifiers

PMID31308803
PMCPMC6606827
OpenAlexW2801638461

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.