Evidence map›Paper›PMID 31295805›Full record

ReviewInternational journal of molecular sciences2019

Protective Effects of Euthyroidism Restoration on Mitochondria Function and Quality Control in Cardiac Pathophysiology.

Francesca Forini, Giuseppina Nicolini, Claudia Kusmic, Giorgio Iervasi

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
2.7field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 49 citations in OpenAlex.

  1. Article
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  12. Review
  13. mtDNA and Mitochondrial Stress Signaling in Human Diseases: A Special Issue.International journal of molecular sciences · 2020
    Article
  14. Review
  15. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Francesca ForiniInstitute of Clinical Physiology, CNR, via G.Moruzzi1, 56124 Pisa, Italy. simona@ifc.cnr.it.
Giuseppina NicoliniInstitute of Clinical Physiology, CNR, via G.Moruzzi1, 56124 Pisa, Italy.
Claudia KusmicInstitute of Clinical Physiology, CNR, via G.Moruzzi1, 56124 Pisa, Italy.ORCID 0000-0002-8064-8311
Giorgio IervasiInstitute of Clinical Physiology, CNR, via G.Moruzzi1, 56124 Pisa, Italy.
Istituto di Fisiologia Clinica · IT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mitochondrial dysfunctions are major contributors to heart disease onset and progression. Under ischemic injuries or cardiac overload, mitochondrial-derived oxidative stress, Ca

Indexed as

AnimalsBiological TransportCalciumDisease SusceptibilityEnergy MetabolismGene Expression RegulationHeart DiseasesHomeostasisHumansMitochondria, HeartMitophagyOxidative StressSignal TransductionThyroid GlandThyroid HormonesCalciumThyroid Hormonescalcium handlingcardiac diseaseinflammationmitochondrial quality controloxidative stressthyroid hormone homeostasis

Identifiers

PMID31295805
PMCPMC6678270
OpenAlexW2960589322

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.