Evidence map›Paper›PMID 31292382›Full record

ArticleInternal medicine (Tokyo, Japan)2019

Laterally Spreading Adenocarcinoma Involving the Lower Bile Duct and Duodenum Expressing Heterogeneous Immunohistochemical Phenotypes.

Katsuyuki Miyabe, Kenji Notohara, Go Asano, Kenta Kachi, Akihisa Kato, Makoto Natsume, Naruomi Jinno, Yasuki Hori, Michihiro Yoshida, Itaru Naitoh and 4 more

Open access · diamondAbstract readCase Reports
In one paragraph

Article in Internal medicine (Tokyo, Japan), 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact, top 89% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 0 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 2 institutions in 1 country.

Katsuyuki MiyabeDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Kenji NotoharaDepartment of Anatomic Pathology, Kurashiki Central Hospital, Japan.
Go AsanoDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Kenta KachiDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Akihisa KatoDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Makoto NatsumeDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Naruomi JinnoDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Yasuki HoriDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Michihiro YoshidaDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Itaru NaitohDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Kazuki HayashiDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Hirotaka OharaDepartment of Community-based Medical Education, Nagoya City University Graduate School of Medical Sciences, Japan.
Satoru TakahashiDepartment of Experimental Pathology and Tumor Biology, Nagoya City University Graduate School of Medical Sciences, Japan.
Hiromi KataokaDepartment of Gastroenterology and Metabolism, Nagoya City University Graduate School of Medical Sciences, Japan.
Nagoya City University · JPKurashiki Central Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

A 70-year-old man was admitted to our hospital due to elevated levels of hepatobiliary and pancreatic enzymes. Computed tomography showed contrast-enhanced mucosal hypertrophy from the duodenal papilla to the distal bile duct. Endoscopic examinations revealed a laterally spreading granular tumor and ampullary swelling. After surgical resection, an examination revealed well-differentiated adenocarcinoma of the ampulla with tubular adenoma spreading from the distal common bile duct to the second part of the duodenum showing both bile duct and duodenal phenotypes. To our knowledge, this is the first case of a tumor spreading from the bile duct to the duodenum that exhibited multiple phenotypes.

Indexed as

AdenocarcinomaAdenomaAgedAmpulla of VaterCommon Bile DuctCommon Bile Duct NeoplasmsDuodenal NeoplasmsDuodenumHumansMaleNeoplasm InvasivenessNeoplasms, Multiple PrimaryTomography, X-Ray Computedampullary adenocarcinomabile ductdifferent phenotypesduodenumlaterally spreading tumor

Identifiers

PMID31292382
PMCPMC6875461
OpenAlexW2959124725

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.