Evidence map›Paper›PMID 31275057›Full record

ReviewMediators of inflammation2019

Noncanonical IFN Signaling, Steroids, and STATs: A Probable Role of V-ATPase.

Howard M Johnson, Ezra Noon-Song, Chulbul M Ahmed

Open access · goldAbstract readReview
In one paragraph

Review in Mediators of inflammation, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.8field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 14 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Review
  5. Article
  6. Article
  7. USP2-Related Cellular Signaling and Consequent Pathophysiological Outcomes.International journal of molecular sciences · 2021
    Review
  8. Review
  9. Review
  10. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Howard M JohnsonDepartment of Microbiology and Cell Science, University of Florida, Gainesville, FL, USA.ORCID 0000-0003-2706-1623
Ezra Noon-SongDepartment of Microbiology and Cell Science, University of Florida, Gainesville, FL, USA.
Chulbul M AhmedDepartment of Microbiology and Cell Science, University of Florida, Gainesville, FL, USA.
University of Florida · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

A small group of only seven transcription factors known as STATs (signal transducer and activator of transcription) are considered to be canonical determinants of specific gene activation for a plethora of ligand/receptor systems. The activation of STATs involves a family of four tyrosine kinases called JAK kinases. JAK1 and JAK2 activate STAT1 in the cytoplasm at the heterodimeric gamma interferon (IFN

Indexed as

AnimalsHumansInterferonsSignal TransductionSTAT2 Transcription FactorSTAT Transcription FactorsSteroidsTYK2 KinaseVacuolar Proton-Translocating ATPasesInterferonsSTAT2 Transcription FactorSTAT Transcription FactorsSteroidsTYK2 KinaseVacuolar Proton-Translocating ATPases

Identifiers

PMID31275057
PMCPMC6558600
OpenAlexW2947296782

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.