Evidence map›Paper›PMID 31269449›Full record

ArticleCell reports2019

Hcr1/eIF3j Is a 60S Ribosomal Subunit Recycling Accessory Factor In Vivo.

David J Young, Nicholas R Guydosh

Abstract read
In one paragraph

Article in Cell reports, 2019. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. RNAViruses · 2026
    Review
  3. Article
  4. Review
  5. Review
  6. Kar4 acts as a Ste12 regulator inbioRxiv : the preprint server for biology · 2025
    Article
  7. Structural insights into the role of eIF3 in translation mediated by the HCV IRES.Proceedings of the National Academy of Sciences of the United States of America · 2025
    Article
  8. Review
  9. Article
  10. Article
  11. Article
  12. Functional Activity of Isoform 2 of Human eRF1.International journal of molecular sciences · 2024
    Article
  13. Article
  14. Article
  15. Article
  16. Review
  17. Article
  18. Article
  19. Review
  20. Rebirth of the translational machinery: The importance of recycling ribosomes.BioEssays : news and reviews in molecular, cellular and developmental biology · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

David J YoungLaboratory of Biochemistry and Genetics, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Bethesda, MD 20892, USA.
Nicholas R GuydoshLaboratory of Biochemistry and Genetics, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Bethesda, MD 20892, USA. Electronic address: nicholas.guydosh@nih.gov.

Funding

Molecular mechanism of the ribosome and functions of translational regulationZIADK075132 · NIDDK · NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES · PI GUYDOSH, NICHOLAS · 2016 to 2025
$14.3M
Intramural NIH HHS ZIA DK075132
6 · The paper itself

Abstract

Hcr1/eIF3j is a sub-stoichiometric subunit of eukaryotic initiation factor 3 (eIF3) that can dissociate the post-termination 40S ribosomal subunit from mRNA in vitro. We examine this ribosome recycling role in vivo by ribosome profiling and reporter assays and find that loss of Hcr1 leads to reinitiation of translation in 3' UTRs, consistent with a defect in recycling. However, the defect appears to be in the recycling of the 60S subunit, rather than the 40S subunit, because reinitiation does not require an AUG codon and is suppressed by overexpression of the 60S dissociation factor Rli1/ABCE1. Consistent with a 60S recycling role, overexpression of Hcr1 cannot compensate for loss of 40S recycling factors Tma64/eIF2D and Tma20/MCT-1. Intriguingly, loss of Hcr1 triggers greater expression of RLI1 via an apparent feedback loop. These findings suggest Hcr1/eIF3j is recruited to ribosomes at stop codons and may coordinate the transition to a new round of translation.

Indexed as

3' Untranslated RegionsATP-Binding Cassette TransportersEukaryotic Initiation Factor-3Gene OntologyPeptide Chain Initiation, TranslationalPeptide Chain Termination, TranslationalPeptide Initiation FactorsRibosome Subunits, Large, EukaryoticRNA-SeqSaccharomyces cerevisiaeSaccharomyces cerevisiae ProteinsUp-Regulation3' Untranslated RegionsATP-Binding Cassette TransportersEukaryotic Initiation Factor-3HCR1 protein, S cerevisiaePeptide Initiation FactorsRLI1 protein, S cerevisiaeSaccharomyces cerevisiae Proteins3′ UTRABCE1DENReIF2DFe-S biogenesisISCUIsu2Lto1MCT-1ORAOV1readthroughreinitiationribosome recyclingRli1terminationTma20Tma22Tma64

Identifiers

PMID31269449
PMCPMC6661068

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.